Cell Migration Inhibition

"Cell Migration Inhibition" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

Phenomenon of cell-mediated immunity measured by in vitro inhibition of the migration or phagocytosis of antigen-stimulated LEUKOCYTES or MACROPHAGES. Specific CELL MIGRATION ASSAYS have been developed to estimate levels of migration inhibitory factors, immune reactivity against tumor-associated antigens, and immunosuppressive effects of infectious microorganisms.

Descriptor ID

D002464

MeSH Number(s)

G04.299.283.337

Concept/Terms

Cell Migration Inhibition

Below are MeSH descriptors whose meaning is more general than "Cell Migration Inhibition".

Biological Sciences [G]
Cell Physiological Phenomena [G04]
Cell Physiological Processes [G04.299]
Cell Movement [G04.299.283]
Cell Migration Inhibition [G04.299.283.337]

Below are MeSH descriptors whose meaning is related to "Cell Migration Inhibition".

Below are MeSH descriptors whose meaning is more specific than "Cell Migration Inhibition".

publications

Timeline | Most Recent

This graph shows the total number of publications written about "Cell Migration Inhibition" by people in Harvard Catalyst Profiles by year, and whether "Cell Migration Inhibition" was a major or minor topic of these publication.

Bar chart showing 38 publications over 14 distinct years, with a maximum of 7 publications in 2006

To see the data from this visualization as text, click here.

Year	Major Topic	Minor Topic	Total
1999	0	1	1
2000	0	3	3
2001	0	1	1
2002	0	5	5
2003	0	5	5
2004	0	3	3
2005	1	4	5
2006	3	4	7
2007	0	2	2
2008	0	1	1
2009	2	0	2
2010	0	1	1
2013	0	1	1
2017	1	0	1

Below are the most recent publications written about "Cell Migration Inhibition" by people in Profiles.

Selectin catch-bonds mechanotransduce integrin activation and neutrophil arrest on inflamed endothelium under shear flow. Blood. 2017 11 09; 130(19):2101-2110.

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Targeting cells in motion: migrating toward improved therapies. Eur J Immunol. 2013 Jun; 43(6):1430-5.

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Predictors of freedom from disease activity in natalizumab treated-patients with multiple sclerosis. J Neurol Sci. 2012 Dec 15; 323(1-2):104-12.

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Emerging roles of endothelial cells in multiple sclerosis pathophysiology and therapy. Neurol Res. 2012 Oct; 34(8):738-45.

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Differing requirements for CCR4, E-selectin, and a4ß1 for the migration of memory CD4 and activated T cells to dermal inflammation. J Immunol. 2012 Jul 01; 189(1):337-46.

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Cutting edge: loss of a4 integrin expression differentially affects the homing of Th1 and Th17 cells. J Immunol. 2011 Dec 15; 187(12):6176-9.

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In vivo-transmigrated human neutrophils are resistant to antiapoptotic stimulation. J Leukoc Biol. 2011 Dec; 90(6):1055-63.

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Apolipoprotein A-II suppressed concanavalin A-induced hepatitis via the inhibition of CD4 T cell function. J Immunol. 2011 Mar 15; 186(6):3410-20.

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Kruppel-like factor 2 is required for trafficking but not quiescence in postactivated T cells. J Immunol. 2011 Jan 15; 186(2):775-83.

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Efalizumab modulates T cell function both in vivo and in vitro. J Dermatol Sci. 2010 Dec; 60(3):159-66.

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