Receptors, Lysosphingolipid
"Receptors, Lysosphingolipid" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A subfamily of lysophospholipid receptors with specificity for LYSOSPHINGOLIPIDS such as sphingosine-1-phosphate and sphingosine phosphorylcholine.
MeSH Number(s)
D12.776.543.750.100.420.500
Concept/Terms
S1P2 Receptor- S1P2 Receptor
- Receptor, S1P2
- H218 Protein
- EDG5 Protein
- G-Protein Coupled Receptor H218
- G Protein Coupled Receptor H218
- AGR16 Protein
- Edg-5 Receptor
- Edg 5 Receptor
- Receptor, Edg-5
Sphingosine-1-Phosphate Receptors- Sphingosine-1-Phosphate Receptors
- Receptors, Sphingosine-1-Phosphate
- Sphingosine 1 Phosphate Receptors
- Sphingosine-1-Phosphate Receptor
- S1P Receptor
- Sphingosine 1-Phosphate Receptor
- 1-Phosphate Receptor, Sphingosine
- Sphingosine 1 Phosphate Receptor
- Edg Receptors
- Receptors, Edg
- Receptor, Sphingosine-1-Phosphate
- Receptor, Sphingosine 1 Phosphate
S1P4 Receptor- S1P4 Receptor
- Receptor, S1P4
- edg-6 Receptor
- Receptor, edg-6
- edg 6 Receptor
S1P5 Receptor- S1P5 Receptor
- Receptor, S1P5
- Nrg-1 Receptor
- Nrg 1 Receptor
- Receptor, Nrg-1
- edg-8 Protein
- Protein, edg-8
- edg 8 Protein
S1P1 Receptor- S1P1 Receptor
- edg-1 Receptor
- Receptor, edg-1
- edg 1 Receptor
- edg-1 Protein
- edg 1 Protein
S1P3 Receptor- S1P3 Receptor
- Receptor, S1P3
- edg-3 Protein
- Protein, edg-3
- edg 3 Protein
Below are MeSH descriptors whose meaning is more general than "Receptors, Lysosphingolipid".
Below are MeSH descriptors whose meaning is more specific than "Receptors, Lysosphingolipid".
This graph shows the total number of publications written about "Receptors, Lysosphingolipid" by people in Harvard Catalyst Profiles by year, and whether "Receptors, Lysosphingolipid" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2004 | 4 | 0 | 4 |
| 2005 | 6 | 1 | 7 |
| 2006 | 2 | 2 | 4 |
| 2007 | 4 | 2 | 6 |
| 2008 | 3 | 2 | 5 |
| 2009 | 4 | 2 | 6 |
| 2010 | 6 | 2 | 8 |
| 2011 | 4 | 2 | 6 |
| 2012 | 5 | 1 | 6 |
| 2013 | 5 | 1 | 6 |
| 2014 | 6 | 1 | 7 |
| 2015 | 4 | 5 | 9 |
| 2016 | 4 | 0 | 4 |
| 2017 | 6 | 2 | 8 |
| 2018 | 3 | 0 | 3 |
| 2021 | 0 | 1 | 1 |
| 2022 | 0 | 1 | 1 |
| 2023 | 0 | 1 | 1 |
| 2024 | 0 | 1 | 1 |
Below are the most recent publications written about "Receptors, Lysosphingolipid" by people in Profiles.
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Designer high-density lipoprotein particles enhance endothelial barrier function and suppress inflammation. Sci Signal. 2024 Feb 20; 17(824):eadg9256.
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Therapeutic activation of endothelial sphingosine-1-phosphate receptor 1 by chaperone-bound S1P suppresses proliferative retinal neovascularization. EMBO Mol Med. 2023 05 08; 15(5):e16645.
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Sphingosine 1-phosphate receptor-targeted therapeutics in rheumatic diseases. Nat Rev Rheumatol. 2022 06; 18(6):335-351.
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CLIC1 and CLIC4 mediate endothelial S1P receptor signaling to facilitate Rac1 and RhoA activity and function. Sci Signal. 2021 04 20; 14(679).
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Sphingosine 1-Phosphate Receptor 1 Signaling Maintains Endothelial Cell Barrier Function and Protects Against Immune Complex-Induced Vascular Injury. Arthritis Rheumatol. 2018 11; 70(11):1879-1889.
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Colonoscopic-Guided Pinch Biopsies in Mice as a Useful Model for Evaluating the Roles of Host and Luminal Factors in Colonic Inflammation. Am J Pathol. 2018 12; 188(12):2811-2825.
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CD4+ T cells are activated in regional lymph nodes and migrate to skin to initiate lymphedema. Nat Commun. 2018 05 17; 9(1):1970.
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S1P4 Regulates Passive Systemic Anaphylaxis in Mice but Is Dispensable for Canonical IgE-Mediated Responses in Mast Cells. Int J Mol Sci. 2018 Apr 25; 19(5).
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The Role of S1PR3 in Protection from Bacterial Sepsis. Am J Respir Crit Care Med. 2017 12 15; 196(12):1500-1502.
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Bioluminescence imaging of G protein-coupled receptor activation in living mice. Nat Commun. 2017 10 27; 8(1):1163.