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Direct screening for chromatin status on DNA barcodes in yeast delineates the regulome of H3K79 methylation by Dot1.
Flexibility in crosstalk between H2B ubiquitination and H3 methylation in vivo.
Dot1 histone methyltransferases share a distributive mechanism but have highly diverged catalytic properties.
The upstreams and downstreams of H3K79 methylation by DOT1L.
Dot1 promotes H2B ubiquitination by a methyltransferase-independent mechanism.
Conserved crosstalk between histone deacetylation and H3K79 methylation generates DOT1L-dose dependency in HDAC1-deficient thymic lymphoma.
The histone methyltransferase DOT1L prevents antigen-independent differentiation and safeguards epigenetic identity of CD8+ T cells.
Histone Lysine N Methyltransferase