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One or more keywords matched the following items that are connected to Campbell, Kevin
Item TypeName
Academic Article Mutations in B3GALNT2 cause congenital muscular dystrophy and hypoglycosylation of a-dystroglycan.
Concept Glycosylation
Academic Article LARGE can functionally bypass alpha-dystroglycan glycosylation defects in distinct congenital muscular dystrophies.
Academic Article Molecular recognition by LARGE is essential for expression of functional dystroglycan.
Academic Article O-mannosyl phosphorylation of alpha-dystroglycan is required for laminin binding.
Academic Article Post-translational disruption of dystroglycan-ligand interactions in congenital muscular dystrophies.
Academic Article Disruption of perlecan binding and matrix assembly by post-translational or genetic disruption of dystroglycan function.
Academic Article Functional glycosylation of dystroglycan is crucial for thymocyte development in the mouse.
Academic Article Like-acetylglucosaminyltransferase (LARGE)-dependent modification of dystroglycan at Thr-317/319 is required for laminin binding and arenavirus infection.
Academic Article Basal lamina strengthens cell membrane integrity via the laminin G domain-binding motif of alpha-dystroglycan.
Academic Article Mouse fukutin deletion impairs dystroglycan processing and recapitulates muscular dystrophy.
Academic Article Residual laminin-binding activity and enhanced dystroglycan glycosylation by LARGE in novel model mice to dystroglycanopathy.
Academic Article Dystroglycan: from biosynthesis to pathogenesis of human disease.
Academic Article Old World arenavirus infection interferes with the expression of functional alpha-dystroglycan in the host cell.
Academic Article The glucuronyltransferase B4GAT1 is required for initiation of LARGE-mediated a-dystroglycan functional glycosylation.
Search Criteria
  • Glycosylation
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.