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One or more keywords matched the following items that are connected to Weil, Brent
Item TypeName
Concept Myocardial Reperfusion Injury
Academic Article Pretreatment with intracoronary mimosine improves postischemic myocardial functional recovery.
Academic Article Systemic pretreatment with dimethyloxalylglycine increases myocardial HIF-1a and VEGF production and improves functional recovery after acute ischemia/reperfusion.
Academic Article Exogenous high-mobility group box 1 improves myocardial recovery after acute global ischemia/reperfusion injury.
Academic Article TGF-a equalizes age disparities in stem cell-mediated cardioprotection.
Academic Article TNF receptor 2, not TNF receptor 1, enhances mesenchymal stem cell-mediated cardiac protection following acute ischemia.
Academic Article Acute postischemic treatment with estrogen receptor-alpha agonist or estrogen receptor-beta agonist improves myocardial recovery.
Academic Article Pretreating mesenchymal stem cells with interleukin-1ß and transforming growth factor-ß synergistically increases vascular endothelial growth factor production and improves mesenchymal stem cell-mediated myocardial protection after acute ischemia.
Academic Article Toll-like receptor 2 mediates mesenchymal stem cell-associated myocardial recovery and VEGF production following acute ischemia-reperfusion injury.
Academic Article Transforming growth factor-alpha does not protect myocardium during acute ischemia/reperfusion.
Academic Article Intracoronary mesenchymal stem cells promote postischemic myocardial functional recovery, decrease inflammation, and reduce apoptosis via a signal transducer and activator of transcription 3 mechanism.
Academic Article Interleukin-10 protects the ischemic heart from reperfusion injury via the STAT3 pathway.
Academic Article Preconditioning mesenchymal stem cells with transforming growth factor-alpha improves mesenchymal stem cell-mediated cardioprotection.
Academic Article Signaling via GPR30 protects the myocardium from ischemia/reperfusion injury.
Academic Article Both endogenous and exogenous testosterone decrease myocardial STAT3 activation and SOCS3 expression after acute ischemia and reperfusion.
Academic Article Estrogen receptor beta mediates increased activation of PI3K/Akt signaling and improved myocardial function in female hearts following acute ischemia.
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  • Myocardial Reperfusion Injury
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.