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Individualized neuropathic pain therapy based on phenotyping: are we there yet?
Short Report: TRPV1-polymorphism 1911 A>G alters capsaicin-induced sensory changes in healthy subjects.
No pain, still gain (of function): the relation between sensory profiles and the presence or absence of self-reported pain in a large multicenter cohort of patients with neuropathy.
The need for previous knowledge does not render quantitative sensory testing a "failure" but part of a larger picture of the relationship between nociception and pain.