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Recombinant vascular endothelial growth factor 121 infusion lowers blood pressure and improves renal function in rats with placentalischemia-induced hypertension.
eNOS deficiency acts through endothelin to aggravate sFlt-1-induced pre-eclampsia-like phenotype.
Hemodynamic, vascular, and reproductive impact of FMS-like tyrosine kinase 1 (FLT1) blockade on the uteroplacental circulation during normal mouse pregnancy.
Soluble endoglin contributes to the pathogenesis of preeclampsia.
Exposure to experimental preeclampsia in mice enhances the vascular response to future injury.
Placental Growth Factor Administration Abolishes Placental Ischemia-Induced Hypertension.
Placental Growth Factor Reduces Blood Pressure in a Uteroplacental Ischemia Model of Preeclampsia in Nonhuman Primates.
Soluble fms-like tyrosine kinase 1 promotes angiotensin II sensitivity in preeclampsia.