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Early complement activation and decreased levels of glycosylphosphatidylinositol-anchored complement inhibitors in human and experimental diabetic retinopathy.
IGF-I mRNA and signaling in the diabetic retina.
Defective myogenic response to posture change in retinal vessels of well-controlled type 1 diabetic patients with no retinopathy.
Bax is increased in the retina of diabetic subjects and is associated with pericyte apoptosis in vivo and in vitro.
Studies of rat and human retinas predict a role for the polyol pathway in human diabetic retinopathy.
Increased prevalence of microthromboses in retinal capillaries of diabetic individuals.
Activation of nuclear factor-kappaB induced by diabetes and high glucose regulates a proapoptotic program in retinal pericytes.
Measurement of gene expression in human retinal microvessels by solution hybridization.
The transforming growth factor-beta pathway is a common target of drugs that prevent experimental diabetic retinopathy.
Response of capillary cell death to aminoguanidine predicts the development of retinopathy: comparison of diabetes and galactosemia.
Fibronectin overexpression in retinal microvessels of patients with diabetes.
Retinal haemodynamics in individuals with well-controlled type 1 diabetes.
Aspirin at low-intermediate concentrations protects retinal vessels in experimental diabetic retinopathy through non-platelet-mediated effects.
Vascular wall von Willebrand factor in human diabetic retinopathy.
Defective Myogenic Response of Retinal Vessels Is Associated With Accelerated Onset of Retinopathy in Type 1 Diabetic Individuals.
HIGH GLUCOSE, DNA, AND DIABETIC COMPLICATIONS
Mechanisms of action of drugs that prevent experimental diabetic retinopathy
The Increased Transforming Growth Factor-ß Signaling Induced by Diabetes Protects Retinal Vessels.