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Protein kinase A signalling via CREB controls myogenesis induced by Wnt proteins.
Mitochondrial cyclic AMP response element-binding protein (CREB) mediates mitochondrial gene expression and neuronal survival.
Serine 133-phosphorylated CREB induces transcription via a cooperative mechanism that may confer specificity to neurotrophin signals.
Trans-synaptic regulation of gene expression.
Coupling of the RAS-MAPK pathway to gene activation by RSK2, a growth factor-regulated CREB kinase.
L-type voltage-sensitive Ca2+ channel activation regulates c-fos transcription at multiple levels.
Cyclic AMP-Dependent Protein Kinases
Cyclic AMP Response Element-Binding Protein
Dynamic regulation of neuronal NO synthase transcription by calcium influx through a CREB family transcription factor-dependent mechanism.
Nerve growth factor-induced neuronal differentiation after dominant repression of both type I and type II cAMP-dependent protein kinase activities.
Mediation by a CREB family transcription factor of NGF-dependent survival of sympathetic neurons.
A dominant-negative inhibitor of CREB reveals that it is a general mediator of stimulus-dependent transcription of c-fos.
SRF mediates activity-induced gene expression and synaptic plasticity but not neuronal viability.
A late phase of cerebellar long-term depression requires activation of CaMKIV and CREB.
An NGF-TrkA-mediated retrograde signal to transcription factor CREB in sympathetic neurons.
Function and regulation of CREB family transcription factors in the nervous system.
A nitric oxide signaling pathway controls CREB-mediated gene expression in neurons.
Apoptosis, axonal growth defects, and degeneration of peripheral neurons in mice lacking CREB.
CREB family transcription factors inhibit neuronal suicide.
What a privilege to reside at the synapse: NMDA receptor signaling to CREB.