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Gene-targeted deletion and replacement mutations of the T-cell receptor beta-chain enhancer: the role of enhancer elements in controlling V(D)J recombination accessibility.
Promotion of V(D)J recombinational accessibility by the intronic E kappa element: role of the kappa B motif.
Mechanisms that direct ordered assembly of T cell receptor beta locus V, D, and J gene segments.
Impaired viability and profound block in thymocyte development in mice lacking the adaptor protein SLP-76.
Extrachromosomal recombination substrates recapitulate beyond 12/23 restricted VDJ recombination in nonlymphoid cells.
E mu N- and E mu L-myc cooperate with E mu pim-1 to generate lymphoid tumors at high frequency in double-transgenic mice.
Separate elements control DJ and VDJ rearrangement in a transgenic recombination substrate.
Dramatically increased rearrangement and peripheral representation of Vbeta14 driven by the 3'Dbeta1 recombination signal sequence.
Productive coupling of accessible Vbeta14 segments and DJbeta complexes determines the frequency of Vbeta14 rearrangement.
Vbeta cluster sequences reduce the frequency of primary Vbeta2 and Vbeta14 rearrangements.
Differential activation of transcription versus recombination of transgenic T cell receptor beta variable region gene segments in B and T lineage cells.
Restriction of endogenous T cell antigen receptor beta rearrangements to Vbeta14 through selective recombination signal sequence modifications.
Fc gamma RII/III and CD2 expression mark distinct subpopulations of immature CD4-CD8- murine thymocytes: in vivo developmental kinetics and T cell receptor beta chain rearrangement status.
Gene Rearrangement, beta-Chain T-Cell Antigen Receptor
Gene Rearrangement beta Chain T Cell Antigen Receptor