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Endometrial cancer is a receptor-mediated target for Mullerian Inhibiting Substance.
Interferon-tau suppresses prostaglandin F2alpha secretion independently of the mitogen-activated protein kinase and nuclear factor kappa B pathways.
Leptin serves as an upstream activator of an obligatory signaling cascade in the embryo-implantation process.
Loss of cables, a cyclin-dependent kinase regulatory protein, is associated with the development of endometrial hyperplasia and endometrial cancer.
Leptin-induced increase in leukemia inhibitory factor and its receptor by human endometrium is partially mediated by interleukin 1 receptor signaling.
Leptin regulation of proangiogenic molecules in benign and cancerous endometrial cells.
BAG-1 expression in normal and neoplastic endometrium.
Stabilizing nuclear p27kip1 as a therapeutic target for endometrial cancer
Cables role in endometrial differentiation and cancer
Overactive mTOR signaling leads to endometrial hyperplasia in aged women and mice.