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Le Gall, Sylvie
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Le Gall, Sylvie
Transmission and accumulation of CTL escape variants drive negative associations between HIV polymorphisms and HLA.
Portable flanking sequences modulate CTL epitope processing.
Mutation of a conserved residue (D123) required for oligomerization of human immunodeficiency virus type 1 Nef protein abolishes interaction with human thioesterase and results in impairment of Nef biological functions.
Antigen processing by nardilysin and thimet oligopeptidase generates cytotoxic T cell epitopes.
Immune selection for altered antigen processing leads to cytotoxic T lymphocyte escape in chronic HIV-1 infection.
Escape from the dominant HLA-B27-restricted cytotoxic T-lymphocyte response in Gag is associated with a dramatic reduction in human immunodeficiency virus type 1 replication.
Selection, transmission, and reversion of an antigen-processing cytotoxic T-lymphocyte escape mutation in human immunodeficiency virus type 1 infection.
Analysis of Nef-induced MHC-I endocytosis.
Nef interacts with the mu subunit of clathrin adaptor complexes and reveals a cryptic sorting signal in MHC I molecules.
The major genetic determinants of HIV-1 control affect HLA class I peptide presentation.
Variable HIV peptide stability in human cytosol is critical to epitope presentation and immune escape.
HIV-1 gag cytotoxic T lymphocyte epitopes vary in presentation kinetics relative to HLA class I downregulation.
A Conserved HIV-1-Derived Peptide Presented by HLA-E Renders Infected T-cells Highly Susceptible to Attack by NKG2A/CD94-Bearing Natural Killer Cells.
Role of antigen processing HIV CTL immunodominance
The HLA-E peptidome in HIV infection
Posttranscriptional Regulation of HLA-A Protein Expression by Alternative Polyadenylation Signals Involving the RNA-Binding Protein Syncrip.
HLA A Antigens