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The worm has turned--microbial virulence modeled in Caenorhabditis elegans.
Contribution of gelatinase, serine protease, and fsr to the pathogenesis of Enterococcus faecalis endophthalmitis.
Caenorhabditis elegans as a host for the study of host-pathogen interactions.
Identification of novel antimicrobials using a live-animal infection model.
High-throughput screen for novel antimicrobials using a whole animal infection model.
The Enterococcus faecalis fsrB gene, a key component of the fsr quorum-sensing system, is associated with virulence in the rabbit endophthalmitis model.
Gram-Positive Bacterial Infections
Both live and dead Enterococci activate Caenorhabditis elegans host defense via immune and stress pathways.
Gram Positive Bacterial Infections