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Impaired glucose tolerance, but not impaired fasting glucose, is associated with increased levels of coronary heart disease risk factors: results from the Baltimore Longitudinal Study on Aging.
Type 2 diabetes-associated missense polymorphisms KCNJ11 E23K and ABCC8 A1369S influence progression to diabetes and response to interventions in the Diabetes Prevention Program.
Risk variable clustering in the insulin resistance syndrome. The Framingham Offspring Study.
Effect of aging on A1C levels in individuals without diabetes: evidence from the Framingham Offspring Study and the National Health and Nutrition Examination Survey 2001-2004.
Metabolic risk factors worsen continuously across the spectrum of nondiabetic glucose tolerance. The Framingham Offspring Study.
Hyperinsulinemia, hyperglycemia, and impaired hemostasis: the Framingham Offspring Study.
The natural history of progression from normal glucose tolerance to type 2 diabetes in the Baltimore Longitudinal Study of Aging.
Using metabolic syndrome traits for efficient detection of impaired glucose tolerance.
The effect of acarbose on insulin sensitivity in subjects with impaired glucose tolerance.
Impaired fasting glucose and impaired glucose tolerance: implications for care.
Navigating the choices for diabetes prevention.
Does diabetes prevention translate into reduced long-term vascular complications of diabetes?