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Targeted disruption of the glucose transporter 4 selectively in muscle causes insulin resistance and glucose intolerance.
Protein-tyrosine phosphatase 1B deficiency reduces insulin resistance and the diabetic phenotype in mice with polygenic insulin resistance.
Adipose tissue selective insulin receptor knockout protects against obesity and obesity-related glucose intolerance.
Deletion of Shp2 tyrosine phosphatase in muscle leads to dilated cardiomyopathy, insulin resistance, and premature death.
IRS-1 transgenic mice show increased epididymal fat mass and insulin resistance.
Specific roles of the p110alpha isoform of phosphatidylinsositol 3-kinase in hepatic insulin signaling and metabolic regulation.
PKCd regulates hepatic insulin sensitivity and hepatosteatosis in mice and humans.
Overexpression of Rad in muscle worsens diet-induced insulin resistance and glucose intolerance and lowers plasma triglyceride level.
beta-cell-specific deletion of the Igf1 receptor leads to hyperinsulinemia and glucose intolerance but does not alter beta-cell mass.
Insulin regulates liver metabolism in vivo in the absence of hepatic Akt and Foxo1.
Tbx15 controls skeletal muscle fibre-type determination and muscle metabolism.
Role of PKCd in Insulin Sensitivity and Skeletal Muscle Metabolism.
Adipocyte Dynamics and Reversible Metabolic Syndrome in Mice with an Inducible Adipocyte-Specific Deletion of the Insulin Receptor.
Endothelial insulin receptors differentially control insulin signaling kinetics in peripheral tissues and brain of mice.
Divergent effects of glucose and fructose on hepatic lipogenesis and insulin signaling.
Insulin signaling in the hippocampus and amygdala regulates metabolism and neurobehavior.
Muscle Specific Insulin Receptor Overexpression Protects Mice from Diet-Induced Glucose Intolerance but Leads to Post-Receptor Insulin Resistance.