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Expression of the elastolytic cathepsins S and K in human atheroma and regulation of their production in smooth muscle cells.
Deficiency of cathepsin S reduces atherosclerosis in LDL receptor-deficient mice.
Cathepsin L expression and regulation in human abdominal aortic aneurysm, atherosclerosis, and vascular cells.
Augmented expression and activity of extracellular matrix-degrading enzymes in regions of low endothelial shear stress colocalize with coronary atheromata with thin fibrous caps in pigs.
Deficiency of the cysteine protease cathepsin S impairs microvessel growth.
Cathepsin L activity is essential to elastase perfusion-induced abdominal aortic aneurysms in mice.
Cathepsin L activity controls adipogenesis and glucose tolerance.
Cathepsin K deficiency reduces elastase perfusion-induced abdominal aortic aneurysms in mice.
Cathepsin L deficiency reduces diet-induced atherosclerosis in low-density lipoprotein receptor-knockout mice.
Deficiency of Cathepsin S reduces atherosclerosis in low-density lipoprotein receptor-deficient mice
Cathepsin S-mediated fibroblast trans-differentiation contributes to left ventricular remodelling after myocardial infarction.
Cathepsin G activity lowers plasma LDL and reduces atherosclerosis.
Cathepsin G deficiency reduces periaortic calcium chloride injury-induced abdominal aortic aneurysms in mice.
Cathepsin S inhibition changes regulatory T-cell activity in regulating bladder cancer and immune cell proliferation and apoptosis.
Cathepsin K Deficiency Ameliorates Systemic Lupus Erythematosus-like Manifestations in Faslpr Mice.
Cathepsin K-deficiency impairs mouse cardiac function after myocardial infarction.