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Facilitation at single synapses probed with optical quantal analysis.
SK channels and NMDA receptors form a Ca2+-mediated feedback loop in dendritic spines.
Fasting activation of AgRP neurons requires NMDA receptors and involves spinogenesis and increased excitatory tone.
Structure and function of dendritic spines.
The life cycle of Ca(2+) ions in dendritic spines.
Regulation of synaptic signalling by postsynaptic, non-glutamate receptor ion channels.
Nonlinear regulation of unitary synaptic signals by CaV(2.3) voltage-sensitive calcium channels located in dendritic spines.
Biphasic synaptic Ca influx arising from compartmentalized electrical signals in dendritic spines.
Distinct structural and ionotropic roles of NMDA receptors in controlling spine and synapse stability.
Neuronal activity regulates diffusion across the neck of dendritic spines.
Ca(2+) signaling in dendritic spines.
Natural oligomers of the Alzheimer amyloid-beta protein induce reversible synapse loss by modulating an NMDA-type glutamate receptor-dependent signaling pathway.
Cholinergic interneurons mediate fast VGluT3-dependent glutamatergic transmission in the striatum.
Cholinergic modulation of multivesicular release regulates striatal synaptic potency and integration.
Competitive regulation of synaptic Ca2+ influx by D2 dopamine and A2A adenosine receptors.
Phosphorylation of Ser1166 on GluN2B by PKA is critical to synaptic NMDA receptor function and Ca2+ signaling in spines.
CRISPR/Cas9-mediated gene knock-down in post-mitotic neurons.
Receptors N Methyl D Aspartate