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Inhibition of interneuron firing extends the spread of endocannabinoid signaling in the cerebellum.
Modulation of transmission during trains at a cerebellar synapse.
Sustained elevation of dendritic calcium evokes widespread endocannabinoid release and suppression of synapses onto cerebellar Purkinje cells.
Prolonged synaptic currents and glutamate spillover at the parallel fiber to stellate cell synapse.
Interaction of postsynaptic receptor saturation with presynaptic mechanisms produces a reliable synapse.
Hyperpolarization induces a long-term increase in the spontaneous firing rate of cerebellar Golgi cells.
Identification of an inhibitory circuit that regulates cerebellar Golgi cell activity.
Noradrenergic control of associative synaptic plasticity by selective modulation of instructive signals.
Calcium microdomains near R-type calcium channels control the induction of presynaptic long-term potentiation at parallel fiber to purkinje cell synapses.
Brief presynaptic bursts evoke synapse-specific retrograde inhibition mediated by endogenous cannabinoids.
Contributions of receptor desensitization and saturation to plasticity at the retinogeniculate synapse.
Cerebellar depolarization-induced suppression of inhibition is mediated by endogenous cannabinoids.
Variance-mean analysis in the presence of a rapid antagonist indicates vesicle depletion underlies depression at the climbing fiber synapse.
Local interneurons regulate synaptic strength by retrograde release of endocannabinoids.
Quantal events shape cerebellar interneuron firing.
The influence of multivesicular release and postsynaptic receptor saturation on transmission at granule cell to Purkinje cell synapses.
Dynamics of fast and slow inhibition from cerebellar golgi cells allow flexible control of synaptic integration.
Somatic spikes regulate dendritic signaling in small neurons in the absence of backpropagating action potentials.
Excitatory Amino Acid Antagonists
Excitatory Amino Acid Agonists
Calcium-dependent PKC isoforms have specialized roles in short-term synaptic plasticity.
Excitatory Amino Acids