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Early acute rejection episodes are reversible following retransplantation into a syngeneic donor and do not progress to chronic rejection.
Quantification of donor-derived DNA in serum: a new approach of acute rejection diagnosis in a rat kidney transplantation model.
Host leukocytes and their products in chronic kidney allograft rejection in rats.
Early cellular markers for the prediction of chronic graft deterioration in a retransplantation model.
Complete reversal of acute rejection episodes after syngeneic retransplantation is associated with a TH1/TH2 shift.
Inhibition of intercellular adhesion molecule-1 with antisense deoxynucleotides prolongs renal isograft survival in the rat.
Nonimmune factors may contribute to chronic rejection of kidney transplants.
Neutrophils and macrophages are prominent in the pathophysiology of chronic rejection of rat kidney allografts.
Tolerance induction by the graft itself.
Alloantigen-independent factors lead to signs of chronic rejection in long-term kidney isografts.
Reversibility of chronic renal allograft rejection. Critical effect of time after transplantation suggests both host immune dependent and independent phases of progressive injury.
Antigen-independent events mimic characteristic changes of chronic allograft rejection.
Long-term kidney isografts develop functional and morphologic changes that mimic those of chronic allograft rejection.