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Formation of the clot.
Thrombin-initiated platelet activation in vivo is vWF independent during thrombus formation in a laser injury model.
Eculizumab therapy results in rapid and sustained decreases in markers of thrombin generation and inflammation in patients with PNH independent of its effects on hemolysis and microparticle formation.
Defective PDI release from platelets and endothelial cells impairs thrombus formation in Hermansky-Pudlak syndrome.
HUMAN MONOCLONAL AUTOANTIBODIES TO PLATELETS
Protein disulfide isomerase inhibition blocks thrombin generation in humans by interfering with platelet factor V activation.
Protein disulfide isomerase regulation by nitric oxide maintains vascular quiescence and controls thrombus formation.