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Neuronal D-serine regulates dendritic architecture in the somatosensory cortex.
Glutamate receptor composition of the post-synaptic density is altered in genetic mouse models of NMDA receptor hypo- and hyperfunction.
The NMDA receptor co-agonists, D-serine and glycine, regulate neuronal dendritic architecture in the somatosensory cortex.
Beyond the dopamine receptor: novel therapeutic targets for treating schizophrenia.
Glutamatergic synaptic dysregulation in schizophrenia: therapeutic implications.
Multiple risk pathways for schizophrenia converge in serine racemase knockout mice, a mouse model of NMDA receptor hypofunction.
D-serine deficiency attenuates the behavioral and cellular effects induced by the hallucinogenic 5-HT(2A) receptor agonist DOI.
Chronic D-serine reverses arc expression and partially rescues dendritic abnormalities in a mouse model of NMDA receptor hypofunction.
Subchronic pharmacological and chronic genetic NMDA receptor hypofunction differentially regulate the Akt signaling pathway and Arc expression in juvenile and adult mice.
The NMDA receptor 'glycine modulatory site' in schizophrenia: D-serine, glycine, and beyond.
[JSNP Excellent Presentation Award for CINP2014].
Neuronal serine racemase regulates extracellular D-serine levels in the adult mouse hippocampus.
History of the Concept of Disconnectivity in Schizophrenia.
An mGlu5-Positive Allosteric Modulator Rescues the Neuroplasticity Deficits in a Genetic Model of NMDA Receptor Hypofunction in Schizophrenia.
The NMDA Receptor and Schizophrenia: From Pathophysiology to Treatment.
The Rise and Fall of the d-Serine-Mediated Gliotransmission Hypothesis.
Enhanced astrocytic d-serine underlies synaptic damage after traumatic brain injury.
Altered CREB Binding to Activity-Dependent Genes in Serine Racemase Deficient Mice, a Mouse Model of Schizophrenia.
Astrocytes in primary cultures express serine racemase, synthesize d-serine and acquire A1 reactive astrocyte features.
Receptors N Methyl D Aspartate