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CDYL bridges REST and histone methyltransferases for gene repression and suppression of cellular transformation.
The Brd4 extraterminal domain confers transcription activation independent of pTEFb by recruiting multiple proteins, including NSD3.
NSD3-NUT fusion oncoprotein in NUT midline carcinoma: implications for a novel oncogenic mechanism.
Histone Lysine N Methyltransferase