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Halofuginone inhibits TH17 cell differentiation by activating the amino acid starvation response.
Halofuginone and other febrifugine derivatives inhibit prolyl-tRNA synthetase.
Characterization of Plasmodium liver stage inhibition by halofuginone.
A genomic and evolutionary approach reveals non-genetic drug resistance in malaria.
The cytoplasmic prolyl-tRNA synthetase of the malaria parasite is a dual-stage target of febrifugine and its analogs.
The Adaptive Proline Response in P. falciparum Is Independent of PfeIK1 and eIF2a Signaling.
Aminoacyl-tRNA synthetase inhibition activates a pathway that branches from the canonical amino acid response in mammalian cells.