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Aldosterone inactivates the endothelin-B receptor via a cysteinyl thiol redox switch to decrease pulmonary endothelial nitric oxide levels and modulate pulmonary arterial hypertension.
Upregulation of steroidogenic acute regulatory protein by hypoxia stimulates aldosterone synthesis in pulmonary artery endothelial cells to promote pulmonary vascular fibrosis.
Characterization of right ventricular remodeling and failure in a chronic pulmonary hypertension model.
Combination proximal pulmonary artery coiling and distal embolization induces chronic elevations in pulmonary artery pressure in Swine.
Emerging Concepts in the Molecular Basis of Pulmonary Arterial Hypertension: Part II: Neurohormonal Signaling Contributes to the Pulmonary Vascular and Right Ventricular Pathophenotype of Pulmonary Arterial Hypertension.
Catheter-based therapies for patients with medication-refractory pulmonary arterial hypertension.
Elevated pulmonary arterial and systemic plasma aldosterone levels associate with impaired cardiac reserve capacity during exercise in left ventricular systolic heart failure patients: A pilot study.
Molecular Mechanisms of Pulmonary Vascular Remodeling in Pulmonary Arterial Hypertension.
Up-regulation of the mammalian target of rapamycin complex 1 subunit Raptor by aldosterone induces abnormal pulmonary artery smooth muscle cell survival patterns to promote pulmonary arterial hypertension.
Intergrated Endothelial Phenotyping to Redefine Pulmonary Hypertension
Catheter-Based Therapy for Inoperable Chronic Thromboembolic Pulmonary Hypertension.
NEDD9 targets COL3A1 to promote endothelial fibrosis and pulmonary arterial hypertension.
A novel secreted-cAMP pathway inhibits pulmonary hypertension via a feed-forward mechanism.
Comprehensive Diagnostic Evaluation of Cardiovascular Physiology in Patients With Pulmonary Vascular Disease: Insights From the PVDOMICS Program.
Hypoxic activation of glucose-6-phosphate dehydrogenase controls the expression of genes involved in the pathogenesis of pulmonary hypertension through the regulation of DNA methylation.