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Genetic determinants of vascular oxidant stress and endothelial dysfunction.
Glutathione peroxidase-1 regulates mitochondrial function to modulate redox-dependent cellular responses.
Glucose-6-phosphate dehydrogenase overexpression decreases endothelial cell oxidant stress and increases bioavailable nitric oxide.
Increased myocardial dysfunction after ischemia-reperfusion in mice lacking glucose-6-phosphate dehydrogenase.
Human alpha B-crystallin mutation causes oxido-reductive stress and protein aggregation cardiomyopathy in mice.
Glucose-6-phosphate dehydrogenase-deficient mice have increased renal oxidative stress and increased albuminuria.
Cyclic strain modulates resistance to oxidant stress by increasing G6PDH expression in smooth muscle cells.
Redox Pioneer: Professor Joseph Loscalzo.
Glutathione peroxidase-1 modulates lipopolysaccharide-induced adhesion molecule expression in endothelial cells by altering CD14 expression.
Glutathione peroxidase-1 deficiency augments proinflammatory cytokine-induced redox signaling and human endothelial cell activation.
Impaired angiogenesis in glutathione peroxidase-1-deficient mice is associated with endothelial progenitor cell dysfunction.
Glutathione peroxidase-3 deficiency promotes platelet-dependent thrombosis in vivo.
G6PD AND OXIDANT STRESS IN VASCULAR SMOOTH MUSCLE CELLS
Ebselen does not improve oxidative stress and vascular function in patients with diabetes: a randomized, crossover trial.