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S100A13 mediates the copper-dependent stress-induced release of IL-1alpha from both human U937 and murine NIH 3T3 cells.
Copper induces the assembly of a multiprotein aggregate implicated in the release of fibroblast growth factor 1 in response to stress.
The alternative translation of synaptotagmin 1 mediates the non-classical release of FGF1.
The non-classical export routes: FGF1 and IL-1alpha point the way.
The non-transmembrane form of Delta1, but not of Jagged1, induces normal migratory behavior accompanied by fibroblast growth factor receptor 1-dependent transformation.
The intracellular translocation of the components of the fibroblast growth factor 1 release complex precedes their assembly prior to export.
Notch activation suppresses fibroblast growth factor-dependent cellular transformation.
A positive FGFR3/FOXN1 feedback loop underlies benign skin keratosis versus squamous cell carcinoma formation in humans.
Sphingosine kinase 1 is a critical component of the copper-dependent FGF1 export pathway.
Receptors, Fibroblast Growth Factor
Receptor, Fibroblast Growth Factor, Type 3
Fibroblast Growth Factor 1
Fibroblast Growth Factor 2
Receptor, Fibroblast Growth Factor, Type 1
Sustained Akt Activity Is Required to Maintain Cell Viability in Seborrheic Keratosis, a Benign Epithelial Tumor.
Fibroblast Growth Factor 3