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Timing of neurologic deterioration in massive middle cerebral artery infarction: a multicenter review.
Distal middle cerebral artery occlusion model for real time optical monitoring of cerebral blood flow and metabolism
Mild induced hypertension improves blood flow and oxygen metabolism in transient focal cerebral ischemia.
Normobaric hyperoxia improves cerebral blood flow and oxygenation, and inhibits peri-infarct depolarizations in experimental focal ischaemia.
Rho-kinase inhibition acutely augments blood flow in focal cerebral ischemia via endothelial mechanisms.
Soluble guanylate cyclase alpha1beta1 limits stroke size and attenuates neurological injury.
Vasoconstrictive neurovascular coupling during focal ischemic depolarizations.
Neuroanatomic correlates of stroke-related myocardial injury.
Infarction, Middle Cerebral Artery
Multiparametric, longitudinal optical coherence tomography imaging reveals acute injury and chronic recovery in experimental ischemic stroke.
Endothelial dysfunction abrogates the efficacy of normobaric hyperoxia in stroke.
Migraine prophylaxis, ischemic depolarizations, and stroke outcomes in mice.
Supply-demand mismatch transients in susceptible peri-infarct hot zones explain the origins of spreading injury depolarizations.
Sustained functional improvement by hepatocyte growth factor-like small molecule BB3 after focal cerebral ischemia in rats and mice.
Enriched Environment Elicits Proangiogenic Mechanisms After Focal Cerebral Ischemia.
Different Effects of Normobaric Oxygen in Normotensive Versus Hypertensive Rats After Focal Cerebral Ischemia.
Multicenter preclinical trial of rho-kinase inhibitor fasudil in acute focal cerebral ischemia and reperfusion
Infarction Middle Cerebral Artery