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Hyperacute lung rejection in the pig-to-human model 4: evidence for complement and antibody independent mechanisms.
Thromboxane mediates pulmonary hypertension and lung inflammation during hyperacute lung rejection.
Thromboxane mediates pulmonary vasoconstriction and contributes to cytotoxicity in pig lungs perfused with fresh human blood.
Hyperacute lung rejection in the pig-to-human model. 2. Synergy between soluble and membrane complement inhibition.