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One or more keywords matched the following items that are connected to Van Raamsdonk, Jeremy
Item TypeName
Concept Caenorhabditis elegans Proteins
Academic Article FUdR causes a twofold increase in the lifespan of the mitochondrial mutant gas-1.
Academic Article Deletion of the mitochondrial superoxide dismutase sod-2 extends lifespan in Caenorhabditis elegans.
Academic Article Uncoupling of oxidative stress resistance and lifespan in long-lived isp-1 mitochondrial mutants in Caenorhabditis elegans.
Academic Article Novel animal model defines genetic contributions for neuron-to-neuron transfer of a-synuclein.
Academic Article Oxidative stress is increased in C. elegans models of Huntington's disease but does not contribute to polyglutamine toxicity phenotypes.
Academic Article Modeling Parkinson's Disease in C. elegans.
Academic Article Mitochondrial and cytoplasmic ROS have opposing effects on lifespan.
Academic Article Activation of DAF-16/FOXO by reactive oxygen species contributes to longevity in long-lived mitochondrial mutants in Caenorhabditis elegans.
Academic Article Activation of the mitochondrial unfolded protein response promotes longevity and dopamine neuron survival in Parkinson's disease models.
Academic Article a-synuclein expression from a single copy transgene increases sensitivity to stress and accelerates neuronal loss in genetic models of Parkinson's disease.
Academic Article Resistance to Stress Can Be Experimentally Dissociated From Longevity.
Academic Article Mitochondrial unfolded protein response transcription factor ATFS-1 promotes longevity in a long-lived mitochondrial mutant through activation of stress response pathways.
Academic Article Disruption of mitochondrial dynamics increases stress resistance through activation of multiple stress response pathways.
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  • Caenorhabditis elegans Proteins
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.