"Trimethyltin Compounds" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Organic compounds composed of tin and three methyl groups. Affect mitochondrial metabolism and inhibit oxidative phosphorylation by acting directly on the energy conserving processes.
Below are MeSH descriptors whose meaning is more general than "Trimethyltin Compounds".
Below are MeSH descriptors whose meaning is more specific than "Trimethyltin Compounds".
This graph shows the total number of publications written about "Trimethyltin Compounds" by people in Harvard Catalyst Profiles by year, and whether "Trimethyltin Compounds" was a major or minor topic of these publication.
![]()
To see the data from this visualization as text,
click here.
Below are the most recent publications written about "Trimethyltin Compounds" by people in Profiles.
-
Lithium promotes neuronal repair and ameliorates depression-like behavior following trimethyltin-induced neuronal loss in the dentate gyrus. PLoS One. 2014; 9(2):e87953.
-
Beneficial in vivo effect of aripiprazole on neuronal regeneration following neuronal loss in the dentate gyrus: evaluation using a mouse model of trimethyltin-induced neuronal loss/self-repair in the dentate gyrus. J Pharmacol Sci. 2014; 124(1):99-111.
-
Opposing roles of glucocorticoid receptor and mineralocorticoid receptor in trimethyltin-induced cytotoxicity in the mouse hippocampus. Neurosci Lett. 2012 Mar 09; 511(2):116-9.
-
[Stimulative effect of edaravone on growth of neuronal stem cells and precursor cells following lesioning of hippocampal dentate gyrus]. Nihon Shinkei Seishin Yakurigaku Zasshi. 2011 Apr; 31(2):99-100.
-
Indomethacin ameliorates trimethyltin-induced neuronal damage in vivo by attenuating oxidative stress in the dentate gyrus of mice. Biol Pharm Bull. 2011; 34(12):1856-63.
-
Neural stem cells modified to express BDNF antagonize trimethyltin-induced neurotoxicity through PI3K/Akt and MAP kinase pathways. J Cell Physiol. 2010 Sep; 224(3):710-21.
-
Enhanced neurogenesis in the olfactory bulb in adult mice after injury induced by acute treatment with trimethyltin. J Neurosci Res. 2010 May 01; 88(6):1242-51.
-
Endogenous and exogenous glucocorticoids prevent trimethyltin from causing neuronal degeneration of the mouse brain in vivo: involvement of oxidative stress pathways. J Pharmacol Sci. 2009 Aug; 110(4):424-36.
-
High susceptibility of cortical neural progenitor cells to trimethyltin toxicity: involvement of both caspases and calpain in cell death. Neurochem Int. 2009 Sep; 55(4):257-64.
-
Activation of c-Jun N-terminal kinase cascades is involved in part of the neuronal degeneration induced by trimethyltin in cortical neurons of mice. J Pharmacol Sci. 2009 Jan; 109(1):60-70.