Muscle Fibers, Slow-Twitch
"Muscle Fibers, Slow-Twitch" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Skeletal muscle fibers characterized by their expression of the Type I MYOSIN HEAVY CHAIN isoforms which have low ATPase activity and effect several other functional properties - shortening velocity, power output, rate of tension redevelopment.
MeSH Number(s)
A10.690.552.500.500.700
A11.620.249.700
Concept/Terms
Muscle Fibers, Slow-Twitch- Muscle Fibers, Slow-Twitch
- Fiber, Slow-Twitch Muscle
- Fibers, Slow-Twitch Muscle
- Muscle Fiber, Slow-Twitch
- Muscle Fibers, Slow Twitch
- Slow-Twitch Muscle Fiber
- Slow-Twitch Muscle Fibers
- Muscle Fibers, Type I
Muscle Fibers, Red- Muscle Fibers, Red
- Fiber, Red Muscle
- Fibers, Red Muscle
- Muscle Fiber, Red
- Red Muscle Fiber
- Red Muscle Fibers
Below are MeSH descriptors whose meaning is more general than "Muscle Fibers, Slow-Twitch".
Below are MeSH descriptors whose meaning is more specific than "Muscle Fibers, Slow-Twitch".
This graph shows the total number of publications written about "Muscle Fibers, Slow-Twitch" by people in Harvard Catalyst Profiles by year, and whether "Muscle Fibers, Slow-Twitch" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1997 | 2 | 0 | 2 |
| 1998 | 1 | 1 | 2 |
| 1999 | 1 | 2 | 3 |
| 2000 | 2 | 1 | 3 |
| 2001 | 1 | 0 | 1 |
| 2002 | 4 | 4 | 8 |
| 2003 | 3 | 1 | 4 |
| 2004 | 2 | 0 | 2 |
| 2006 | 0 | 1 | 1 |
| 2007 | 1 | 1 | 2 |
| 2010 | 2 | 0 | 2 |
| 2011 | 1 | 2 | 3 |
| 2013 | 1 | 1 | 2 |
| 2014 | 1 | 0 | 1 |
| 2015 | 1 | 0 | 1 |
| 2016 | 0 | 1 | 1 |
| 2019 | 1 | 0 | 1 |
| 2021 | 0 | 1 | 1 |
Below are the most recent publications written about "Muscle Fibers, Slow-Twitch" by people in Profiles.
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Cancer-induced muscle atrophy is determined by intrinsic muscle oxidative capacity. FASEB J. 2021 07; 35(7):e21714.
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A Global Loss of Dio2 Leads to Unexpected Changes in Function and Fiber Types of Slow Skeletal Muscle in Male Mice. Endocrinology. 2019 05 01; 160(5):1205-1222.
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Effect of Eukarion-134 on Akt-mTOR signalling in the rat soleus during 7 days of mechanical unloading. Exp Physiol. 2018 04 01; 103(4):545-558.
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Recessive mutations in the kinase ZAK cause a congenital myopathy with fibre type disproportion. Brain. 2017 01; 140(1):37-48.
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Muscle dysfunction in a zebrafish model of Duchenne muscular dystrophy. Physiol Genomics. 2016 11 01; 48(11):850-860.
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Muscle weakness in TPM3-myopathy is due to reduced Ca2+-sensitivity and impaired acto-myosin cross-bridge cycling in slow fibres. Hum Mol Genet. 2015 Nov 15; 24(22):6278-92.
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Differentiation of the intracellular structure of slow- versus fast-twitch muscle fibers through evaluation of the dielectric properties of tissue. Phys Med Biol. 2014 May 21; 59(10):2369-80.
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A randomized study on the effect of vitamin D3 supplementation on skeletal muscle morphology and vitamin D receptor concentration in older women. J Clin Endocrinol Metab. 2013 Dec; 98(12):E1927-35.
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Nonsurgically induced disuse muscle atrophy and neuromuscular dysfunction upregulates alpha7 acetylcholine receptors. Can J Physiol Pharmacol. 2014 Jan; 92(1):1-8.
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NFAT isoforms regulate muscle fiber type transition without altering CaN during aerobic training. Int J Sports Med. 2013 Oct; 34(10):861-7.