Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)

"Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus, MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure, which enables searching at various levels of specificity.

MeSH information

Definition | Details | More General Concepts | Related Concepts | More Specific Concepts

An enzyme that catalyzes the synthesis of fructose-6-phosphate plus GLUTAMINE from GLUTAMATE plus glucosamine-6-phosphate.

Descriptor ID

D005945

MeSH Number(s)

D08.811.913.477.700.500

Concept/Terms

Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)

Below are MeSH descriptors whose meaning is more general than "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)".

Chemicals and Drugs [D]
Enzymes and Coenzymes [D08]
Enzymes [D08.811]
Transferases [D08.811.913]
Nitrogenous Group Transferases [D08.811.913.477]
Transaminases [D08.811.913.477.700]
Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing) [D08.811.913.477.700.500]

Below are MeSH descriptors whose meaning is related to "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)".

Below are MeSH descriptors whose meaning is more specific than "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)".

publications

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This graph shows the total number of publications written about "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)" by people in Harvard Catalyst Profiles by year, and whether "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)" was a major or minor topic of these publication.

Below are the most recent publications written about "Glutamine-Fructose-6-Phosphate Transaminase (Isomerizing)" by people in Profiles.

Parasitoid wasp venom elevates sorbitol and alters expression of metabolic genes in human kidney cells. Toxicon. 2019 Apr 01; 161:57-64.

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Congenital Myasthenic Syndrome: Spectrum of Mutations in an Indian Cohort. J Clin Neuromuscul Dis. 2018 Sep; 20(1):14-27.

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Azide- and Alkyne-Bearing Metabolic Chemical Reporters of Glycosylation Show Structure-Dependent Feedback Inhibition of the Hexosamine Biosynthetic Pathway. Chembiochem. 2018 09 17; 19(18):1918-1921.

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2-Deoxy-d-glucose increases GFAT1 phosphorylation resulting in endoplasmic reticulum-related apoptosis via disruption of protein N-glycosylation in pancreatic cancer cells. Biochem Biophys Res Commun. 2018 06 27; 501(3):668-673.

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A Systems-Level Analysis of the Peripheral Nerve Intrinsic Axonal Growth Program. Neuron. 2016 Mar 02; 89(5):956-70.

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Mutations in GFPT1 that underlie limb-girdle congenital myasthenic syndrome result in reduced cell-surface expression of muscle AChR. Hum Mol Genet. 2013 Jul 15; 22(14):2905-13.

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GlmS and NagB regulate amino sugar metabolism in opposing directions and affect Streptococcus mutans virulence. PLoS One. 2012; 7(3):e33382.

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Cardiac O-GlcNAc signaling is increased in hypertrophy and heart failure. Physiol Genomics. 2012 Feb 01; 44(2):162-72.

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A head-to-head comparison of eneamide and epoxyamide inhibitors of glucosamine-6-phosphate synthase from the dapdiamide biosynthetic pathway. Biochemistry. 2011 May 17; 50(19):3859-61.

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AMP-activated protein kinase phosphorylates glutamine : fructose-6-phosphate amidotransferase 1 at Ser243 to modulate its enzymatic activity. Genes Cells. 2009 Feb; 14(2):179-89.

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