Large-Conductance Calcium-Activated Potassium Channel beta Subunits
"Large-Conductance Calcium-Activated Potassium Channel beta Subunits" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
The regulatory subunits of large-conductance calcium-activated potassium channels.
MeSH Number(s)
D12.776.157.530.400.600.150.500.750
D12.776.543.550.425.750.150.500.750
D12.776.543.585.400.750.150.500.500
Concept/Terms
Large-Conductance Calcium-Activated Potassium Channel beta Subunits- Large-Conductance Calcium-Activated Potassium Channel beta Subunits
- Large Conductance Calcium Activated Potassium Channel beta Subunits
- Large-Conductance Calcium-Activated Potassium Channels, beta Subunits
- Large Conductance Calcium Activated Potassium Channels, beta Subunits
- MaxiK Channel beta Subunits
Below are MeSH descriptors whose meaning is more general than "Large-Conductance Calcium-Activated Potassium Channel beta Subunits".
Below are MeSH descriptors whose meaning is more specific than "Large-Conductance Calcium-Activated Potassium Channel beta Subunits".
This graph shows the total number of publications written about "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" by people in Harvard Catalyst Profiles by year, and whether "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
Year | Major Topic | Minor Topic | Total |
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2003 | 0 | 1 | 1 |
2008 | 1 | 0 | 1 |
2009 | 1 | 0 | 1 |
2012 | 2 | 0 | 2 |
2014 | 1 | 0 | 1 |
Below are the most recent publications written about "Large-Conductance Calcium-Activated Potassium Channel beta Subunits" by people in Profiles.
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Relation between BK-a/ß4-mediated potassium secretion and ENaC-mediated sodium reabsorption. Kidney Int. 2014 Jul; 86(1):139-45.
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Flow-sensitive K+-coupled ATP secretion modulates activity of the epithelial Na+ channel in the distal nephron. J Biol Chem. 2012 Nov 09; 287(46):38552-8.
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Bicarbonate promotes BK-a/ß4-mediated K excretion in the renal distal nephron. Am J Physiol Renal Physiol. 2012 Dec 01; 303(11):F1563-71.
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Common charge-shift mutation Glu65Lys in K+ channel ß1-Subunit KCNMB1: pleiotropic consequences for glomerular filtration rate and progressive renal disease. Am J Nephrol. 2010; 32(5):414-24.
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The KCNMB1 E65K variant is associated with reduced central pulse pressure in the community-based Framingham Offspring Cohort. J Hypertens. 2009 Jan; 27(1):55-60.
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Structural basis for toxin resistance of beta4-associated calcium-activated potassium (BK) channels. J Biol Chem. 2008 Aug 29; 283(35):24177-84.
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Lysine-rich extracellular rings formed by hbeta2 subunits confer the outward rectification of BK channels. PLoS One. 2008 May 07; 3(5):e2114.
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The protective effect of KCNMB1 E65K against hypertension is restricted to blood pressure treatment with beta-blockade. J Hum Hypertens. 2008 Jul; 22(7):512-5.
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Structural determinants for functional coupling between the beta and alpha subunits in the Ca2+-activated K+ (BK) channel. J Gen Physiol. 2006 Feb; 127(2):191-204.
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Effect of 17beta-estradiol on mRNA expression of large- conductance, voltage-dependent, and calcium-activated potassium channel alpha and beta subunits in guinea pig. Endocrine. 2003 Apr; 20(3):227-37.