Fanconi Anemia Complementation Group G Protein
"Fanconi Anemia Complementation Group G Protein" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A Fanconi anemia complementation group protein that undergoes PHOSPHORYLATION by CDC2 PROTEIN KINASE during MITOSIS. It forms a complex with other FANCONI ANEMIA PROTEINS and helps protect CELLS from DNA DAMAGE by genotoxic agents.
MeSH Number(s)
D12.776.313.906
D12.776.744.488
Concept/Terms
Fanconi Anemia Complementation Group G Protein- Fanconi Anemia Complementation Group G Protein
- XRCC9 Protein
- Fanconi Anemia Group G Protein
- X-Ray Repair Complementing Defective Repair In Chinese Hamster Cells 9 Protein
- X Ray Repair Complementing Defective Repair In Chinese Hamster Cells 9 Protein
- FANCG Protein
- Fanconi Anemia Group G Complementing Protein
Below are MeSH descriptors whose meaning is more general than "Fanconi Anemia Complementation Group G Protein".
Below are MeSH descriptors whose meaning is more specific than "Fanconi Anemia Complementation Group G Protein".
This graph shows the total number of publications written about "Fanconi Anemia Complementation Group G Protein" by people in Harvard Catalyst Profiles by year, and whether "Fanconi Anemia Complementation Group G Protein" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1999 | 0 | 1 | 1 |
| 2000 | 0 | 3 | 3 |
| 2001 | 0 | 4 | 4 |
| 2002 | 0 | 1 | 1 |
| 2003 | 0 | 1 | 1 |
| 2006 | 0 | 1 | 1 |
| 2007 | 1 | 0 | 1 |
| 2018 | 1 | 1 | 2 |
Below are the most recent publications written about "Fanconi Anemia Complementation Group G Protein" by people in Profiles.
-
Functional analysis of Fanconi anemia mutations in China. Exp Hematol. 2018 10; 66:32-41.e8.
-
Amelioration of Head and Neck Radiation-Induced Mucositis and Distant Marrow Suppression in Fanca-/- and Fancg-/- Mice by Intraoral Administration of GS-Nitroxide (JP4-039). Radiat Res. 2018 06; 189(6):560-578.
-
Fanconi anemia pathway-deficient tumor cells are hypersensitive to inhibition of ataxia telangiectasia mutated. J Clin Invest. 2007 May; 117(5):1440-9.
-
Regulation of monoubiquitinated PCNA by DUB autocleavage. Nat Cell Biol. 2006 Apr; 8(4):339-47.
-
Acquired FANCA dysfunction and cytogenetic instability in adult acute myelogenous leukemia. Blood. 2003 Jul 01; 102(1):7-16.
-
Heterogeneous activation of the Fanconi anemia pathway by patient-derived FANCA mutants. Hum Mol Genet. 2002 Dec 01; 11(25):3125-34.
-
The Chinese hamster FANCG/XRCC9 mutant NM3 fails to express the monoubiquitinated form of the FANCD2 protein, is hypersensitive to a range of DNA damaging agents and exhibits a normal level of spontaneous sister chromatid exchange. Carcinogenesis. 2001 Dec; 22(12):1939-46.
-
Targeted disruption of the murine Fanconi anemia gene, Fancg/Xrcc9. Blood. 2001 Dec 01; 98(12):3435-40.
-
Function of the Fanconi anemia pathway in Fanconi anemia complementation group F and D1 cells. Exp Hematol. 2001 Dec; 29(12):1448-55.
-
Functional analysis of patient-derived mutations in the Fanconi anemia gene, FANCG/XRCC9. Exp Hematol. 2001 Jul; 29(7):842-9.