Nuclear Receptor Subfamily 1, Group F, Member 3
"Nuclear Receptor Subfamily 1, Group F, Member 3" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
An orphan nuclear receptor found in the THYMUS where it plays a role in regulating the development and maturation of thymocytes. An isoform of this protein, referred to as RORgammaT, is produced by an alternatively transcribed mRNA.
MeSH Number(s)
D12.776.260.698.209.182
D12.776.826.209.182
D12.776.930.669.209.182
Concept/Terms
Nuclear Receptor Subfamily 1, Group F, Member 3- Nuclear Receptor Subfamily 1, Group F, Member 3
- Nuclear Receptor RZR-gamma
- Nuclear Receptor RZR gamma
- Receptor RZR-gamma, Nuclear
- Orphan Nuclear Receptor NR1F3
- RAR-related Orphan Receptor C
- RAR related Orphan Receptor C
- ROR-C Receptors
- ROR C Receptors
- ROR-gamma
- Nuclear Receptor NR1F3
- Receptor NR1F3, Nuclear
- Orphan Nuclear Receptor ROR-gamma
- Orphan Nuclear Receptor ROR gamma
Below are MeSH descriptors whose meaning is more general than "Nuclear Receptor Subfamily 1, Group F, Member 3".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- DNA-Binding Proteins [D12.776.260]
- Receptors, Cytoplasmic and Nuclear [D12.776.260.698]
- Orphan Nuclear Receptors [D12.776.260.698.209]
- Nuclear Receptor Subfamily 1, Group F, Member 3 [D12.776.260.698.209.182]
- Receptors, Cytoplasmic and Nuclear [D12.776.826]
- Orphan Nuclear Receptors [D12.776.826.209]
- Nuclear Receptor Subfamily 1, Group F, Member 3 [D12.776.826.209.182]
- Transcription Factors [D12.776.930]
- Receptors, Cytoplasmic and Nuclear [D12.776.930.669]
- Orphan Nuclear Receptors [D12.776.930.669.209]
- Nuclear Receptor Subfamily 1, Group F, Member 3 [D12.776.930.669.209.182]
Below are MeSH descriptors whose meaning is more specific than "Nuclear Receptor Subfamily 1, Group F, Member 3".
This graph shows the total number of publications written about "Nuclear Receptor Subfamily 1, Group F, Member 3" by people in Harvard Catalyst Profiles by year, and whether "Nuclear Receptor Subfamily 1, Group F, Member 3" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2007 | 0 | 1 | 1 |
| 2008 | 0 | 4 | 4 |
| 2009 | 0 | 2 | 2 |
| 2010 | 1 | 0 | 1 |
| 2011 | 1 | 0 | 1 |
| 2012 | 3 | 4 | 7 |
| 2013 | 0 | 3 | 3 |
| 2014 | 4 | 0 | 4 |
| 2015 | 3 | 5 | 8 |
| 2016 | 1 | 6 | 7 |
| 2017 | 0 | 4 | 4 |
| 2018 | 0 | 2 | 2 |
| 2019 | 3 | 5 | 8 |
| 2020 | 1 | 6 | 7 |
| 2021 | 3 | 2 | 5 |
| 2022 | 4 | 3 | 7 |
| 2023 | 0 | 3 | 3 |
| 2024 | 0 | 1 | 1 |
Below are the most recent publications written about "Nuclear Receptor Subfamily 1, Group F, Member 3" by people in Profiles.
-
Suppressor T helper type 17 cell responses in intestinal transplant recipients with allograft rejection. Hum Immunol. 2024 May; 85(3):110773.
-
A distinct human cell type expressing MHCII and ROR?t with dual characteristics of dendritic cells and type 3 innate lymphoid cells. Proc Natl Acad Sci U S A. 2023 Dec 26; 120(52):e2318710120.
-
The gut microbiota promotes distal tissue regeneration via ROR?+ regulatory T cell emissaries. Immunity. 2023 04 11; 56(4):829-846.e8.
-
Retinoic Acid Receptor-Related Orphan Receptors (RORs) in Eye Development and Disease. Adv Exp Med Biol. 2023; 1415:327-332.
-
An embarrassment of riches: ROR?t+ antigen-presenting cells in peripheral tolerance. Immunity. 2022 11 08; 55(11):1978-1980.
-
Ornithine decarboxylase supports ILC3 responses in infectious and autoimmune colitis through positive regulation of IL-22 transcription. Proc Natl Acad Sci U S A. 2022 Nov 08; 119(45):e2214900119.
-
Mining the microbiota to identify gut commensals modulating neuroinflammation in a mouse model of multiple sclerosis. Microbiome. 2022 10 17; 10(1):174.
-
Sequestration of gut pathobionts in intraluminal casts, a mechanism to avoid dysregulated T cell activation by pathobionts. Proc Natl Acad Sci U S A. 2022 10 11; 119(41):e2209624119.
-
Selenoprotein I deficiency in T cells promotes differentiation into tolerant phenotypes while decreasing Th17 pathology. J Leukoc Biol. 2022 12; 112(6):1387-1397.
-
Autoreactive memory Th17 cells are principally derived from T-bet+ROR?t+ Th17/1 effectors. J Autoimmun. 2022 05; 129:102816.