This page shows the publications Kiyoshi Itagaki has written about Receptors, Formyl Peptide.
Formyl Peptide Receptor-1 Blockade Prevents Receptor Regulation by Mitochondrial Danger-Associated Molecular Patterns and Preserves Neutrophil Function After Trauma. Crit Care Med. 2020 02; 48(2):e123-e132.
Mitochondrial damage associated molecular patterns from femoral reamings activate neutrophils through formyl peptide receptors and P44/42 MAP kinase. J Orthop Trauma. 2010 Sep; 24(9):534-8.
A subset of five human mitochondrial formyl peptides mimics bacterial peptides and functionally deactivates human neutrophils. J Trauma Acute Care Surg. 2018 11; 85(5):936-943.
Mitochondrial damage-associated molecular patterns from fractures suppress pulmonary immune responses via formyl peptide receptors 1 and 2. J Trauma Acute Care Surg. 2015 Feb; 78(2):272-9; discussion 279-81.
Mitochondrial peptides are potent immune activators that activate human neutrophils via FPR-1. J Trauma. 2010 Jun; 68(6):1328-32; discussion 1332-4.
Circulating mitochondrial DAMPs cause inflammatory responses to injury. Nature. 2010 Mar 04; 464(7285):104-7.
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