Harvard Catalyst Profiles

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Susan Bonner-Weir, Ph.D.

Co-Author

This page shows the publications co-authored by Susan Bonner-Weir and Cristina Aguayo-Mazzucato.
Connection Strength

6.871
  1. Acceleration of ß Cell Aging Determines Diabetes and Senolysis Improves Disease Outcomes. Cell Metab. 2019 07 02; 30(1):129-142.e4.
    View in: PubMed
    Score: 0.853
  2. T3 Induces Both Markers of Maturation and Aging in Pancreatic ß-Cells. Diabetes. 2018 07; 67(7):1322-1331.
    View in: PubMed
    Score: 0.788
  3. Pancreatic ß Cell Regeneration as a Possible Therapy for Diabetes. Cell Metab. 2018 01 09; 27(1):57-67.
    View in: PubMed
    Score: 0.757
  4. ß Cell Aging Markers Have Heterogeneous Distribution and Are Induced by Insulin Resistance. Cell Metab. 2017 Apr 04; 25(4):898-910.e5.
    View in: PubMed
    Score: 0.735
  5. Physiology: Pancreatic ß-cell heterogeneity revisited. Nature. 2016 07 21; 535(7612):365-6.
    View in: PubMed
    Score: 0.699
  6. MAFA and T3 Drive Maturation of Both Fetal Human Islets and Insulin-Producing Cells Differentiated From hESC. J Clin Endocrinol Metab. 2015 Oct; 100(10):3651-9.
    View in: PubMed
    Score: 0.653
  7. Thyroid hormone promotes postnatal rat pancreatic ß-cell development and glucose-responsive insulin secretion through MAFA. Diabetes. 2013 May; 62(5):1569-80.
    View in: PubMed
    Score: 0.548
  8. Mafa expression enhances glucose-responsive insulin secretion in neonatal rat beta cells. Diabetologia. 2011 Mar; 54(3):583-93.
    View in: PubMed
    Score: 0.476
  9. Stem cell therapy for type 1 diabetes mellitus. Nat Rev Endocrinol. 2010 Mar; 6(3):139-48.
    View in: PubMed
    Score: 0.449
  10. Unique Human and Mouse ß-Cell Senescence-Associated Secretory Phenotype (SASP) Reveal Conserved Signaling Pathways and Heterogeneous Factors. Diabetes. 2021 05; 70(5):1098-1116.
    View in: PubMed
    Score: 0.241
  11. Dynamic development of the pancreas from birth to adulthood. Ups J Med Sci. 2016 May; 121(2):155-8.
    View in: PubMed
    Score: 0.171
  12. ß-cell dedifferentiation in diabetes is important, but what is it? Islets. 2013 Sep-Dec; 5(5):233-7.
    View in: PubMed
    Score: 0.146
  13. PDX1 in ducts is not required for postnatal formation of ß-cells but is necessary for their subsequent maturation. Diabetes. 2013 Oct; 62(10):3459-68.
    View in: PubMed
    Score: 0.141
  14. Rat neonatal beta cells lack the specialised metabolic phenotype of mature beta cells. Diabetologia. 2011 Mar; 54(3):594-604.
    View in: PubMed
    Score: 0.119
  15. Subpopulations of GFP-marked mouse pancreatic ß-cells differ in size, granularity, and insulin secretion. Endocrinology. 2012 Nov; 153(11):5180-7.
    View in: PubMed
    Score: 0.033
  16. Mice with a targeted deletion of the type 2 deiodinase are insulin resistant and susceptible to diet induced obesity. PLoS One. 2011; 6(6):e20832.
    View in: PubMed
    Score: 0.031
  17. Tissue-specific disallowance of housekeeping genes: the other face of cell differentiation. Genome Res. 2011 Jan; 21(1):95-105.
    View in: PubMed
    Score: 0.030
Connection Strength
The connection strength for co-authors is the sum of the scores for each of their shared publications.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.