This page shows the publications co-authored by Carl Hauser and Leo Otterbein.
Danger signals from mitochondrial DAMPS in trauma and post-injury sepsis. Eur J Trauma Emerg Surg. 2018 Jun; 44(3):317-324.
Trauma-induced heme release increases susceptibility to bacterial infection. JCI Insight. 2021 Sep 14.
Monocyte exocytosis of mitochondrial danger-associated molecular patterns in sepsis suppresses neutrophil chemotaxis. J Trauma Acute Care Surg. 2021 01 01; 90(1):46-53.
Multiplexed Plasma Immune Mediator Signatures Can Differentiate Sepsis From NonInfective SIRS: American Surgical Association 2020 Annual Meeting Paper. Ann Surg. 2020 10; 272(4):604-610.
Formyl Peptide Receptor-1 Blockade Prevents Receptor Regulation by Mitochondrial Danger-Associated Molecular Patterns and Preserves Neutrophil Function After Trauma. Crit Care Med. 2020 02; 48(2):e123-e132.
Formyl Peptide Receptor-1 Blockade Prevents Receptor Regulation by Mitochondrial Danger-Associated Molecular Patterns and Preserves Neutrophil Function After Trauma. Crit Care Med. 2019 Nov 15.
Carbon monoxide protects the kidney through the central circadian clock and CD39. Proc Natl Acad Sci U S A. 2018 03 06; 115(10):E2302-E2310.
Intratracheal instillation of neutrophils rescues bacterial overgrowth initiated by trauma damage-associated molecular patterns. J Trauma Acute Care Surg. 2017 05; 82(5):853-860.
Mitochondrial DAMPs Are Released During Cardiopulmonary Bypass Surgery and Are Associated With Postoperative Atrial Fibrillation. Heart Lung Circ. 2018 Jan; 27(1):122-129.
Heme as a danger molecule in pathogen recognition. Free Radic Biol Med. 2015 Dec; 89:651-61.
Mitochondrial DNA released by trauma induces neutrophil extracellular traps. PLoS One. 2015; 10(3):e0120549.
Mitochondrial damage-associated molecular patterns from fractures suppress pulmonary immune responses via formyl peptide receptors 1 and 2. J Trauma Acute Care Surg. 2015 Feb; 78(2):272-9; discussion 279-81.
Direct Airway Instillation of Neutrophils Overcomes Chemotactic Deficits Induced by Injury. Shock. 2021 07 01; 56(1):119-124.
Circulating Factors in Trauma Plasma Activate Specific Human Immune Cell Subsets. Injury. 2020 Apr; 51(4):819-829.
A subset of five human mitochondrial formyl peptides mimics bacterial peptides and functionally deactivates human neutrophils. J Trauma Acute Care Surg. 2018 11; 85(5):936-943.
Mitochondrial DAMPs increase endothelial permeability through neutrophil dependent and independent pathways. PLoS One. 2013; 8(3):e59989.
The connection strength for co-authors is the sum of the scores for each of their shared publications.
Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.