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W. Allan Walker, M.D.

Concepts

This page shows the publications W. Walker has written about Toll-Like Receptor 4.
Connection Strength

1.903
  1. The symbiotic bacterial surface factor polysaccharide A on Bacteroides fragilis inhibits IL-1ß-induced inflammation in human fetal enterocytes via toll receptors 2 and 4. PLoS One. 2017; 12(3):e0172738.
    View in: PubMed
    Score: 0.503
  2. Anti-inflammatory effects of Bifidobacterium longum subsp infantis secretions on fetal human enterocytes are mediated by TLR-4 receptors. Am J Physiol Gastrointest Liver Physiol. 2016 10 01; 311(4):G744-G753.
    View in: PubMed
    Score: 0.484
  3. Toll-like receptor-4 in human and mouse colonic epithelium is developmentally regulated: a possible role in necrotizing enterocolitis. Pediatr Res. 2015 Mar; 77(3):416-24.
    View in: PubMed
    Score: 0.431
  4. Lipopolysaccharide-induced human enterocyte tolerance to cytokine-mediated interleukin-8 production may occur independently of TLR-4/MD-2 signaling. Pediatr Res. 2006 Jan; 59(1):89-95.
    View in: PubMed
    Score: 0.230
  5. The developmentally regulated fetal enterocyte gene, ZP4, mediates anti-inflammation by the symbiotic bacterial surface factor polysaccharide A on Bacteroides fragilis. Am J Physiol Gastrointest Liver Physiol. 2019 10 01; 317(4):G398-G407.
    View in: PubMed
    Score: 0.148
  6. The mechanism of excessive intestinal inflammation in necrotizing enterocolitis: an immature innate immune response. PLoS One. 2011 Mar 21; 6(3):e17776.
    View in: PubMed
    Score: 0.083
  7. Probiotics prevent necrotizing enterocolitis by modulating enterocyte genes that regulate innate immune-mediated inflammation. Am J Physiol Gastrointest Liver Physiol. 2013 Jan 15; 304(2):G132-41.
    View in: PubMed
    Score: 0.023
Connection Strength

The connection strength for concepts is the sum of the scores for each matching publication.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.

Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.