Harvard Catalyst Profiles

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John Butler Mulliken, M.D.

Co-Author

This page shows the publications co-authored by John Mulliken and Matthew Warman.
Connection Strength

1.251
  1. Somatic Activating Mutations in GNAQ and GNA11 Are Associated with Congenital Hemangioma. Am J Hum Genet. 2016 06 02; 98(6):1271.
    View in: PubMed
    Score: 0.172
  2. Somatic Activating Mutations in GNAQ and GNA11 Are Associated with Congenital Hemangioma. Am J Hum Genet. 2016 Apr 07; 98(4):789-95.
    View in: PubMed
    Score: 0.170
  3. Molecular genetics and craniofacial surgery. Plast Reconstr Surg. 1996 Mar; 97(3):666-75.
    View in: PubMed
    Score: 0.169
  4. Somatic mosaic activating mutations in PIK3CA cause CLOVES syndrome. Am J Hum Genet. 2012 Jun 08; 90(6):1108-15.
    View in: PubMed
    Score: 0.130
  5. Intramuscular fast-flow vascular anomaly contains somatic MAP2K1 and KRAS mutations. Angiogenesis. 2019 11; 22(4):547-552.
    View in: PubMed
    Score: 0.054
  6. Somatic PIK3CA mutations are present in multiple tissues of facial infiltrating lipomatosis. Pediatr Res. 2017 Nov; 82(5):850-854.
    View in: PubMed
    Score: 0.047
  7. Hemangioma in twins. Ann Plast Surg. 1997 Mar; 38(3):269-74.
    View in: PubMed
    Score: 0.045
  8. Somatic MAP2K1 Mutations Are Associated with Extracranial Arteriovenous Malformation. Am J Hum Genet. 2017 Mar 02; 100(3):546-554.
    View in: PubMed
    Score: 0.045
  9. Endothelial Cells from Capillary Malformations Are Enriched for Somatic GNAQ Mutations. Plast Reconstr Surg. 2016 Jan; 137(1):77e-82e.
    View in: PubMed
    Score: 0.042
  10. Facial Infiltrating Lipomatosis Contains Somatic PIK3CA Mutations in Multiple Tissues. Plast Reconstr Surg. 2015 Oct; 136(4 Suppl):72-3.
    View in: PubMed
    Score: 0.041
  11. A gene for familial venous malformations maps to chromosome 9p in a second large kindred. J Med Genet. 1995 Mar; 32(3):197-9.
    View in: PubMed
    Score: 0.039
  12. A somatic MAP3K3 mutation is associated with verrucous venous malformation. Am J Hum Genet. 2015 Mar 05; 96(3):480-6.
    View in: PubMed
    Score: 0.039
  13. Lymphatic and other vascular malformative/overgrowth disorders are caused by somatic mutations in PIK3CA. J Pediatr. 2015 Apr; 166(4):1048-54.e1-5.
    View in: PubMed
    Score: 0.039
  14. Assignment of a locus for dominantly inherited venous malformations to chromosome 9p. Hum Mol Genet. 1994 Sep; 3(9):1583-7.
    View in: PubMed
    Score: 0.038
  15. PIK3CA activating mutations in facial infiltrating lipomatosis. Plast Reconstr Surg. 2014 Jan; 133(1):12e-19e.
    View in: PubMed
    Score: 0.036
  16. A mutation in the homeodomain of the human MSX2 gene in a family affected with autosomal dominant craniosynostosis. Cell. 1993 Nov 05; 75(3):443-50.
    View in: PubMed
    Score: 0.036
  17. Newly recognized autosomal dominant disorder with craniosynostosis. Am J Med Genet. 1993 Jun 01; 46(4):444-9.
    View in: PubMed
    Score: 0.035
  18. Mutations in a novel factor, glomulin, are responsible for glomuvenous malformations ("glomangiomas"). Am J Hum Genet. 2002 Apr; 70(4):866-74.
    View in: PubMed
    Score: 0.016
  19. Splicing mutations of 54-bp exons in the COL11A1 gene cause Marshall syndrome, but other mutations cause overlapping Marshall/Stickler phenotypes. Am J Hum Genet. 1999 Oct; 65(4):974-83.
    View in: PubMed
    Score: 0.014
  20. A gene for inherited cutaneous venous anomalies ("glomangiomas") localizes to chromosome 1p21-22. Am J Hum Genet. 1999 Jul; 65(1):125-33.
    View in: PubMed
    Score: 0.013
  21. Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2. Cell. 1996 Dec 27; 87(7):1181-90.
    View in: PubMed
    Score: 0.011
  22. Genetic mapping of cleidocranial dysplasia and evidence of a microdeletion in one family. Hum Mol Genet. 1995 Jan; 4(1):71-5.
    View in: PubMed
    Score: 0.010
  23. Assignment of a gene locus involved in craniosynostosis to chromosome 5qter. Hum Mol Genet. 1993 Feb; 2(2):119-22.
    View in: PubMed
    Score: 0.009
Connection Strength
The connection strength for co-authors is the sum of the scores for each of their shared publications.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.