Harvard Catalyst Profiles

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Frederick Jack Schoen, M.D., Ph.D.

Co-Author

This page shows the publications co-authored by Frederick Schoen and Peter Libby.
Connection Strength

0.914
  1. Cardiac transplant graft arteriosclerosis. Trends Cardiovasc Med. 1991 Jul-Aug; 1(5):216-23.
    View in: PubMed
    Score: 0.123
  2. Arteriosclerosis in transplanted hearts: too much and too soon. Can J Cardiol. 1991 Apr; 7(3):XI-XII.
    View in: PubMed
    Score: 0.121
  3. Lipid lowering reduces oxidative stress and endothelial cell activation in rabbit atheroma. Circulation. 2002 Sep 10; 106(11):1390-6.
    View in: PubMed
    Score: 0.067
  4. Activated interstitial myofibroblasts express catabolic enzymes and mediate matrix remodeling in myxomatous heart valves. Circulation. 2001 Nov 20; 104(21):2525-32.
    View in: PubMed
    Score: 0.063
  5. An HMG-CoA reductase inhibitor, cerivastatin, suppresses growth of macrophages expressing matrix metalloproteinases and tissue factor in vivo and in vitro. Circulation. 2001 Jan 16; 103(2):276-83.
    View in: PubMed
    Score: 0.060
  6. Evidence for increased collagenolysis by interstitial collagenases-1 and -3 in vulnerable human atheromatous plaques. Circulation. 1999 May 18; 99(19):2503-9.
    View in: PubMed
    Score: 0.053
  7. Lipid lowering promotes accumulation of mature smooth muscle cells expressing smooth muscle myosin heavy chain isoforms in rabbit atheroma. Circ Res. 1998 Nov 16; 83(10):1015-26.
    View in: PubMed
    Score: 0.051
  8. Nanoparticles targeting extra domain B of fibronectin-specific to the atherosclerotic lesion types III, IV, and V-enhance plaque detection and cargo delivery. Theranostics. 2018; 8(21):6008-6024.
    View in: PubMed
    Score: 0.051
  9. Circumferential stress and matrix metalloproteinase 1 in human coronary atherosclerosis. Implications for plaque rupture. Arterioscler Thromb Vasc Biol. 1996 Aug; 16(8):1070-3.
    View in: PubMed
    Score: 0.044
  10. Smooth muscle cells of the coronary arterial tunica media express tumor necrosis factor-alpha and proliferate during acute rejection of rabbit cardiac allografts. Am J Pathol. 1995 Sep; 147(3):617-26.
    View in: PubMed
    Score: 0.041
  11. Acute rejection accelerates graft coronary disease in transplanted rabbit hearts. Circulation. 1995 Aug 15; 92(4):987-93.
    View in: PubMed
    Score: 0.041
  12. Endothelial and smooth muscle cells express leukocyte adhesion molecules heterogeneously during acute rejection of rabbit cardiac allografts. Am J Pathol. 1994 May; 144(5):938-51.
    View in: PubMed
    Score: 0.037
  13. Immunopathology of coronary arteriosclerosis in transplanted hearts. J Heart Lung Transplant. 1992 May-Jun; 11(3 Pt 2):S5-6.
    View in: PubMed
    Score: 0.033
  14. Human coronary transplantation-associated arteriosclerosis. Evidence for a chronic immune reaction to activated graft endothelial cells. Am J Pathol. 1991 Apr; 138(4):791-8.
    View in: PubMed
    Score: 0.030
  15. Functions of vascular wall cells related to development of transplantation-associated coronary arteriosclerosis. Transplant Proc. 1989 Aug; 21(4):3677-84.
    View in: PubMed
    Score: 0.027
  16. Characterization of human atherosclerotic plaques by intravascular magnetic resonance imaging. Circulation. 2005 Oct 11; 112(15):2324-31.
    View in: PubMed
    Score: 0.021
  17. Genetically determined resistance to collagenase action augments interstitial collagen accumulation in atherosclerotic plaques. Circulation. 2004 Oct 05; 110(14):1953-9.
    View in: PubMed
    Score: 0.019
  18. Selective matrix metalloproteinase inhibition reduces left ventricular remodeling but does not inhibit angiogenesis after myocardial infarction. Circulation. 2002 Feb 12; 105(6):753-8.
    View in: PubMed
    Score: 0.016
  19. Targeted deletion of matrix metalloproteinase-9 attenuates left ventricular enlargement and collagen accumulation after experimental myocardial infarction. J Clin Invest. 2000 Jul; 106(1):55-62.
    View in: PubMed
    Score: 0.014
Connection Strength
The connection strength for co-authors is the sum of the scores for each of their shared publications.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.