Harvard Catalyst Profiles

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William Gerald Austen Jr., M.D.

Co-Author

This page shows the publications co-authored by William Austen and Francis Moore.
Connection Strength

1.194
  1. Murine hindlimb reperfusion injury can be initiated by a self-reactive monoclonal IgM. Surgery. 2004 Aug; 136(2):401-6.
    View in: PubMed
    Score: 0.304
  2. The role of complement and natural antibody in intestinal ischemia-reperfusion injury. Int J Immunopathol Pharmacol. 2003 Jan-Apr; 16(1):1-8.
    View in: PubMed
    Score: 0.272
  3. Improved survival of murine island skin flaps by prevention of reperfusion injury. Plast Reconstr Surg. 2009 May; 123(5):1431-1439.
    View in: PubMed
    Score: 0.106
  4. The differing roles of the classical and mannose-binding lectin complement pathways in the events following skeletal muscle ischemia-reperfusion. J Immunol. 2006 Dec 01; 177(11):8080-5.
    View in: PubMed
    Score: 0.089
  5. IgM binding to injured tissue precedes complement activation during skeletal muscle ischemia-reperfusion. J Surg Res. 2004 Nov; 122(1):29-35.
    View in: PubMed
    Score: 0.077
  6. Reperfusion injury to skeletal muscle affects primarily type II muscle fibers. J Surg Res. 2004 Nov; 122(1):54-60.
    View in: PubMed
    Score: 0.077
  7. Intestinal ischemia-reperfusion injury is mediated by the membrane attack complex. Surgery. 1999 Aug; 126(2):343-8.
    View in: PubMed
    Score: 0.054
  8. Mast cell protease 5 mediates ischemia-reperfusion injury of mouse skeletal muscle. J Immunol. 2005 Jun 01; 174(11):7285-91.
    View in: PubMed
    Score: 0.020
  9. Identification of a specific self-reactive IgM antibody that initiates intestinal ischemia/reperfusion injury. Proc Natl Acad Sci U S A. 2004 Mar 16; 101(11):3886-91.
    View in: PubMed
    Score: 0.018
  10. Functional activity of natural antibody is altered in Cr2-deficient mice. J Immunol. 2002 Nov 15; 169(10):5433-40.
    View in: PubMed
    Score: 0.017
  11. Sialyl Lewis(x) hybridized complement receptor type 1 moderates acid aspiration injury. . 2001 Dec; 281(6):L1494-9.
    View in: PubMed
    Score: 0.016
  12. Mast cells mediate complement activation after acid aspiration. Shock. 2001 Jul; 16(1):21-4.
    View in: PubMed
    Score: 0.015
  13. Moderation of skeletal muscle reperfusion injury by a sLe(x)-glycosylated complement inhibitory protein. . 2001 Jul; 281(1):C224-30.
    View in: PubMed
    Score: 0.015
  14. Recombinant soluble P-selectin glycoprotein ligand 1 moderates local and remote injuries following experimental lower-torso ischaemia. Br J Surg. 2001 Jun; 88(6):825-30.
    View in: PubMed
    Score: 0.015
  15. Soluble P-selectin moderates complement dependent injury. Shock. 2000 Dec; 14(6):610-5.
    View in: PubMed
    Score: 0.015
  16. Soluble P-selectin moderates complement-dependent reperfusion injury of ischemic skeletal muscle. . 2000 Aug; 279(2):C520-8.
    View in: PubMed
    Score: 0.014
  17. Endothelial selectin blockade attenuates lung permeability of experimental acid aspiration. Surgery. 2000 Aug; 128(2):327-31.
    View in: PubMed
    Score: 0.014
  18. The role of complement receptors CD21/CD35 in positive selection of B-1 cells. Curr Top Microbiol Immunol. 2000; 252:57-65.
    View in: PubMed
    Score: 0.014
  19. Neutrophil mediated remote organ injury after lower torso ischemia and reperfusion is selectin and complement dependent. J Trauma. 2000 Jan; 48(1):32-8.
    View in: PubMed
    Score: 0.014
  20. Skeletal muscle reperfusion injury is mediated by neutrophils and the complement membrane attack complex. Am J Physiol. 1999 12; 277(6):C1263-8.
    View in: PubMed
    Score: 0.014
  21. Membrane attack complex of complement and neutrophils mediate the injury of acid aspiration. J Appl Physiol (1985). 1999 Dec; 87(6):2357-61.
    View in: PubMed
    Score: 0.014
Connection Strength
The connection strength for co-authors is the sum of the scores for each of their shared publications.

Publication scores are based on many factors, including how long ago they were written and whether the person is a first or senior author.
Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.