Receptors, Interleukin-17
"Receptors, Interleukin-17" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Cell surface receptors for INTERLEUKIN-17. Several subtypes of receptors have been found, each with its own in specificity for interleukin-17 subtype.
MeSH Number(s)
D12.776.543.750.705.852.420.820
D23.050.301.264.035.820
D23.101.100.110.820
Concept/Terms
Receptors, Interleukin-17- Receptors, Interleukin-17
- Receptors, Interleukin 17
- IL-17 Receptors
- IL 17 Receptors
- Receptors, IL-17
- Interleukin-17 Receptors
- Interleukin 17 Receptors
Interleukin-17 Receptor A- Interleukin-17 Receptor A
- Interleukin 17 Receptor A
- Interleukin-17 Receptor
- Interleukin 17 Receptor
- Receptor, Interleukin-17
- Cdw217 Antigens
- Antigens, CDw217
- CDw217 Antigen
- Antigen, CDw217
Below are MeSH descriptors whose meaning is more general than "Receptors, Interleukin-17".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, Immunologic [D12.776.543.750.705]
- Receptors, Cytokine [D12.776.543.750.705.852]
- Receptors, Interleukin [D12.776.543.750.705.852.420]
- Receptors, Interleukin-17 [D12.776.543.750.705.852.420.820]
- Biological Factors [D23]
- Antigens [D23.050]
- Antigens, Surface [D23.050.301]
- Antigens, Differentiation [D23.050.301.264]
- Antigens, CD [D23.050.301.264.035]
- Receptors, Interleukin-17 [D23.050.301.264.035.820]
- Biological Markers [D23.101]
- Antigens, Differentiation [D23.101.100]
- Antigens, CD [D23.101.100.110]
- Receptors, Interleukin-17 [D23.101.100.110.820]
Below are MeSH descriptors whose meaning is more specific than "Receptors, Interleukin-17".
This graph shows the total number of publications written about "Receptors, Interleukin-17" by people in Harvard Catalyst Profiles by year, and whether "Receptors, Interleukin-17" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2006 | 0 | 2 | 2 |
| 2007 | 1 | 1 | 2 |
| 2008 | 0 | 2 | 2 |
| 2009 | 1 | 2 | 3 |
| 2010 | 3 | 2 | 5 |
| 2011 | 3 | 2 | 5 |
| 2012 | 0 | 4 | 4 |
| 2013 | 1 | 3 | 4 |
| 2014 | 2 | 3 | 5 |
| 2015 | 1 | 5 | 6 |
| 2016 | 2 | 1 | 3 |
| 2017 | 2 | 1 | 3 |
| 2018 | 0 | 1 | 1 |
| 2019 | 0 | 1 | 1 |
| 2020 | 1 | 1 | 2 |
| 2022 | 1 | 0 | 1 |
Below are the most recent publications written about "Receptors, Interleukin-17" by people in Profiles.
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IL-17RA-signaling in Lgr5+ intestinal stem cells induces expression of transcription factor ATOH1 to promote secretory cell lineage commitment. Immunity. 2022 02 08; 55(2):237-253.e8.
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Contribution of a European-Prevalent Variant near CD83 and an East Asian-Prevalent Variant near IL17RB to Herpes Zoster Risk in Tofacitinib Treatment: Results of Genome-Wide Association Study Meta-Analyses. Arthritis Rheumatol. 2021 07; 73(7):1155-1166.
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Systematic analysis of the IL-17 receptor signalosome reveals a robust regulatory feedback loop. EMBO J. 2020 09 01; 39(17):e104202.
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Whole exome sequencing analyses reveal gene-microbiota interactions in the context of IBD. Gut. 2021 02; 70(2):285-296.
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Breast Cancer Index and prediction of benefit from extended endocrine therapy in breast cancer patients treated in the Adjuvant Tamoxifen-To Offer More? (aTTom) trial. Ann Oncol. 2019 11 01; 30(11):1776-1783.
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Act1 is a negative regulator in T and B cells via direct inhibition of STAT3. Nat Commun. 2018 07 16; 9(1):2745.
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Smoking-induced aggravation of experimental arthritis is dependent of aryl hydrocarbon receptor activation in Th17 cells. Arthritis Res Ther. 2018 06 08; 20(1):119.
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IL-17-receptor-associated adaptor Act1 directly stabilizes mRNAs to mediate IL-17 inflammatory signaling. Nat Immunol. 2018 04; 19(4):354-365.
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IL-17RB enhances thyroid cancer cell invasion and metastasis via ERK1/2 pathway-mediated MMP-9 expression. Mol Immunol. 2017 10; 90:126-135.
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Thermoneutral housing exacerbates nonalcoholic fatty liver disease in mice and allows for sex-independent disease modeling. Nat Med. 2017 Jul; 23(7):829-838.