"Gene Knock-In Techniques" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
Techniques used to add in exogenous gene sequence such as mutated genes; REPORTER GENES, to study mechanisms of gene expression; or regulatory control sequences, to study effects of temporal changes to GENE EXPRESSION.
Concept/Terms
Gene Knock-In Techniques- Gene Knock-In Techniques
- Gene Knock-In Technique
- Knock-In Technique, Gene
- Knock-In Techniques, Gene
- Technique, Gene Knock-In
- Techniques, Gene Knock-In
- Gene Knock In Techniques
- Gene Knock-In
- Gene Knock-Ins
- Knock-In, Gene
- Knock-Ins, Gene
- Gene Knock In
- Gene Knock Ins
- In, Gene Knock
- Ins, Gene Knock
- Knock In, Gene
- Knock Ins, Gene
Below are MeSH descriptors whose meaning is more general than "Gene Knock-In Techniques".
Below are MeSH descriptors whose meaning is more specific than "Gene Knock-In Techniques".
This graph shows the total number of publications written about "Gene Knock-In Techniques" by people in Harvard Catalyst Profiles by year, and whether "Gene Knock-In Techniques" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 2008 | 0 | 5 | 5 |
| 2009 | 3 | 13 | 16 |
| 2010 | 1 | 16 | 17 |
| 2011 | 2 | 27 | 29 |
| 2012 | 2 | 11 | 13 |
| 2013 | 1 | 17 | 18 |
| 2014 | 1 | 18 | 19 |
| 2015 | 4 | 16 | 20 |
| 2016 | 1 | 20 | 21 |
| 2017 | 0 | 20 | 20 |
| 2018 | 1 | 13 | 14 |
| 2019 | 2 | 23 | 25 |
| 2020 | 1 | 21 | 22 |
| 2021 | 0 | 18 | 18 |
| 2022 | 0 | 1 | 1 |
Below are the most recent publications written about "Gene Knock-In Techniques" by people in Profiles.
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Proteomic analysis identifies ZMYM2 as endogenous binding partner of TBX18 protein in 293 and A549 cells. Biochem J. 2022 01 14; 479(1):91-109.
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Knockin mouse models demonstrate differential contributions of synaptotagmin-1 and -2 as receptors for botulinum neurotoxins. PLoS Pathog. 2021 10; 17(10):e1009994.
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A Human Pleiotropic Multiorgan Condition Caused by Deficient Wnt Secretion. N Engl J Med. 2021 09 30; 385(14):1292-1301.
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Postnatal Sox6 Regulates Synaptic Function of Cortical Parvalbumin-Expressing Neurons. J Neurosci. 2021 10 27; 41(43):8876-8886.
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Neurovascular imaging with QUTE-CE MRI in APOE4 rats reveals early vascular abnormalities. PLoS One. 2021; 16(8):e0256749.
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Surfactant protein C mutation links postnatal type 2 cell dysfunction to adult disease. JCI Insight. 2021 07 22; 6(14).
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Presynaptic Short-Term Plasticity Persists in the Absence of PKC Phosphorylation of Munc18-1. J Neurosci. 2021 09 01; 41(35):7329-7339.
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Oligomerization of Lrrk controls actin severing and a-synuclein neurotoxicity in vivo. Mol Neurodegener. 2021 05 24; 16(1):33.
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Cytoplasmic FUS triggers early behavioral alterations linked to cortical neuronal hyperactivity and inhibitory synaptic defects. Nat Commun. 2021 05 21; 12(1):3028.
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BAD regulates mammary gland morphogenesis by 4E-BP1-mediated control of localized translation in mouse and human models. Nat Commun. 2021 05 19; 12(1):2939.