Harvard Catalyst Profiles

Contact, publication, and social network information about Harvard faculty and fellows.

Overexpression of Estrogen Receptor Alfa (ER-Alfa)Inhibits the Negative Feedback Regulation of Cholesterol (Ch) Biosynthesis and Increases Newly Synthesized Ch Leading to Biliary Ch Hypersecretion

Overexpression of Estrogen Receptor Alfa (ER-Alfa)Inhibits the Negative Feedback Regulation of Cholesterol (Ch) Biosynthesis and Increases Newly Synthesized Ch Leading to Biliary Ch Hypersecretion. Gastroenterology. 2004; 126(4):A-673.


Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.