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Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice.

Defects in nuclear structure and function promote dilated cardiomyopathy in lamin A/C-deficient mice. J Clin Invest. 2004 Feb; 113(3):357-69.

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Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.