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Cardiac-Specific Overexpression of NO Synthase 3 (NOS3) Attenuates Production of Reactive Oxygen Species and Preserves Cardiac Myocyte Contractile Function During Endotoxemia

Cardiac-Specific Overexpression of NO Synthase 3 (NOS3) Attenuates Production of Reactive Oxygen Species and Preserves Cardiac Myocyte Contractile Function During Endotoxemia. Circulation. 2005; 112:1358.


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Funded by the NIH National Center for Advancing Translational Sciences through its Clinical and Translational Science Awards Program, grant number UL1TR002541.