Maf Transcription Factors, Large
"Maf Transcription Factors, Large" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of high molecular weight Maf transcription factors that contain distinct activation domains.
MeSH Number(s)
D12.776.260.108.500.061
D12.776.930.127.500.061
Below are MeSH descriptors whose meaning is more general than "Maf Transcription Factors, Large".
Below are MeSH descriptors whose meaning is more specific than "Maf Transcription Factors, Large".
This graph shows the total number of publications written about "Maf Transcription Factors, Large" by people in Harvard Catalyst Profiles by year, and whether "Maf Transcription Factors, Large" was a major or minor topic of these publication.
To see the data from this visualization as text,
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Year | Major Topic | Minor Topic | Total |
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2006 | 1 | 0 | 1 |
2008 | 0 | 1 | 1 |
2009 | 2 | 0 | 2 |
2010 | 1 | 0 | 1 |
2013 | 2 | 0 | 2 |
2015 | 1 | 1 | 2 |
2018 | 0 | 1 | 1 |
2019 | 0 | 1 | 1 |
Below are the most recent publications written about "Maf Transcription Factors, Large" by people in Profiles.
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Diabetes relief in mice by glucose-sensing insulin-secreting human a-cells. Nature. 2019 03; 567(7746):43-48.
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T3 Induces Both Markers of Maturation and Aging in Pancreatic ß-Cells. Diabetes. 2018 07; 67(7):1322-1331.
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Compensatory Response by Late Embryonic Tubular Epithelium to the Reduction in Pancreatic Progenitors. PLoS One. 2015; 10(11):e0142286.
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MAFA and T3 Drive Maturation of Both Fetal Human Islets and Insulin-Producing Cells Differentiated From hESC. J Clin Endocrinol Metab. 2015 Oct; 100(10):3651-9.
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In vivo reprogramming of pancreatic acinar cells to three islet endocrine subtypes. Elife. 2014 Jan 01; 3:e01846.
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Reprogramming of various cell types to a beta-like state by Pdx1, Ngn3 and MafA. PLoS One. 2013; 8(11):e82424.
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Differentiation of pancreatic endocrine progenitors reversibly blocked by premature induction of MafA. Dev Biol. 2014 Jan 01; 385(1):2-12.
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Stage specific reprogramming of mouse embryo liver cells to a beta cell-like phenotype. Mech Dev. 2013 Nov-Dec; 130(11-12):602-12.
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PDX1 in ducts is not required for postnatal formation of ß-cells but is necessary for their subsequent maturation. Diabetes. 2013 Oct; 62(10):3459-68.
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Preventing p38 MAPK-mediated MafA degradation ameliorates ß-cell dysfunction under oxidative stress. Mol Endocrinol. 2013 Jul; 27(7):1078-90.