Receptor, Anaphylatoxin C5a
"Receptor, Anaphylatoxin C5a" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A G-protein-coupled receptor that signals an increase in intracellular calcium in response to the potent ANAPHYLATOXIN peptide COMPLEMENT C5A.
MeSH Number(s)
D12.776.543.750.100.024
D12.776.543.750.705.833.550
D23.050.301.264.035.595
D23.101.100.110.595
Concept/Terms
Receptor, Anaphylatoxin C5a- Receptor, Anaphylatoxin C5a
- Antigens, CD88
- C5a Receptors
- Receptors, C5a
- CD88 Antigens
- Complement 5a Receptor
- Receptor, Complement 5a
- Anaphylatoxin C5a Receptor
- CD88 Antigen
- Antigen, CD88
Below are MeSH descriptors whose meaning is more general than "Receptor, Anaphylatoxin C5a".
- Chemicals and Drugs [D]
- Amino Acids, Peptides, and Proteins [D12]
- Proteins [D12.776]
- Membrane Proteins [D12.776.543]
- Receptors, Cell Surface [D12.776.543.750]
- Receptors, G-Protein-Coupled [D12.776.543.750.100]
- Receptor, Anaphylatoxin C5a [D12.776.543.750.100.024]
- Receptors, Immunologic [D12.776.543.750.705]
- Receptors, Complement [D12.776.543.750.705.833]
- Receptor, Anaphylatoxin C5a [D12.776.543.750.705.833.550]
- Biological Factors [D23]
- Antigens [D23.050]
- Antigens, Surface [D23.050.301]
- Antigens, Differentiation [D23.050.301.264]
- Antigens, CD [D23.050.301.264.035]
- Receptor, Anaphylatoxin C5a [D23.050.301.264.035.595]
- Biological Markers [D23.101]
- Antigens, Differentiation [D23.101.100]
- Antigens, CD [D23.101.100.110]
- Receptor, Anaphylatoxin C5a [D23.101.100.110.595]
Below are MeSH descriptors whose meaning is more specific than "Receptor, Anaphylatoxin C5a".
This graph shows the total number of publications written about "Receptor, Anaphylatoxin C5a" by people in Harvard Catalyst Profiles by year, and whether "Receptor, Anaphylatoxin C5a" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1994 | 0 | 2 | 2 |
| 1995 | 0 | 1 | 1 |
| 1996 | 0 | 2 | 2 |
| 1997 | 0 | 1 | 1 |
| 1998 | 0 | 1 | 1 |
| 2000 | 0 | 2 | 2 |
| 2001 | 0 | 2 | 2 |
| 2002 | 0 | 3 | 3 |
| 2003 | 0 | 2 | 2 |
| 2004 | 0 | 1 | 1 |
| 2005 | 1 | 2 | 3 |
| 2007 | 0 | 2 | 2 |
| 2008 | 1 | 1 | 2 |
| 2009 | 1 | 0 | 1 |
| 2010 | 2 | 3 | 5 |
| 2011 | 2 | 0 | 2 |
| 2012 | 2 | 0 | 2 |
| 2013 | 3 | 1 | 4 |
| 2014 | 1 | 0 | 1 |
| 2015 | 1 | 0 | 1 |
| 2016 | 0 | 1 | 1 |
| 2018 | 4 | 0 | 4 |
| 2019 | 3 | 0 | 3 |
| 2021 | 0 | 2 | 2 |
Below are the most recent publications written about "Receptor, Anaphylatoxin C5a" by people in Profiles.
-
Human Neutrophils Respond to Complement Activation and Inhibition in Microfluidic Devices. Front Immunol. 2021; 12:777932.
-
Dynamic regulation of B cell complement signaling is integral to germinal center responses. Nat Immunol. 2021 06; 22(6):757-768.
-
Follicular B2 Cell Activation and Class Switch Recombination Depend on Autocrine C3ar1/C5ar1 Signaling in B2 Cells. J Immunol. 2019 07 15; 203(2):379-388.
-
Atypical complement receptor C5aR2 transports C5a to initiate neutrophil adhesion and inflammation. Sci Immunol. 2019 05 10; 4(35).
-
Pathogenic roles of anti-C1q antibodies in recurrent pregnancy loss. Clin Immunol. 2019 06; 203:37-44.
-
Deletion of Biliverdin Reductase A in Myeloid Cells Promotes Chemokine Expression and Chemotaxis in Part via a Complement C5a--C5aR1 Pathway. J Immunol. 2019 05 15; 202(10):2982-2990.
-
C5aR1 regulates T follicular helper differentiation and chronic graft-versus-host disease bronchiolitis obliterans. JCI Insight. 2018 12 20; 3(24).
-
Blood clotting and traumatic injury with shock mediates complement-dependent neutrophil priming for extracellular ROS, ROS-dependent organ injury and coagulopathy. Clin Exp Immunol. 2018 10; 194(1):103-117.
-
The critical role of C5a as an initiator of neutrophil-mediated autoimmune inflammation of the joint and skin. Semin Immunol. 2018 06; 37:21-29.
-
Complement Receptor C5aR1 Plays an Evolutionarily Conserved Role in Successful Cardiac Regeneration. Circulation. 2018 05 15; 137(20):2152-2165.