GTP-Binding Protein alpha Subunits, Gs
"GTP-Binding Protein alpha Subunits, Gs" is a descriptor in the National Library of Medicine's controlled vocabulary thesaurus,
MeSH (Medical Subject Headings). Descriptors are arranged in a hierarchical structure,
which enables searching at various levels of specificity.
A family of heterotrimeric GTP-binding protein alpha subunits that activate ADENYLATE CYCLASE.
MeSH Number(s)
D08.811.277.040.330.300.200.100.400
D12.644.360.375.100.400
D12.776.157.325.332.100.400
D12.776.476.375.100.400
D12.776.543.325.100.400
Concept/Terms
GTP-Binding Protein alpha Subunits, Gs- GTP-Binding Protein alpha Subunits, Gs
- GTP Binding Protein alpha Subunits, Gs
- G(s), alpha Subunit
- G(s)alpha
- G-Protein, Gs alpha Family
- G Protein, Gs alpha Family
- Stimulatory GTP-Binding Protein, alpha Subunit
- Stimulatory GTP Binding Protein, alpha Subunit
- Gs alpha GTP-Binding Protein Subunits
- Gs alpha GTP Binding Protein Subunits
- Gs Stimulatory G-Proteins
- G-Proteins, Gs Stimulatory
- Gs Stimulatory G Proteins
- Stimulatory G-Proteins, Gs
- Gs, Stimulatory G-Protein
- G-Protein Gs, Stimulatory
- Gs, Stimulatory G Protein
- Stimulatory G-Protein Gs
- Ns Protein, Regulatory
- Regulatory Ns Protein
- alpha-Gs
- alpha Gs
- G-Protein, Stimulatory Gs
- G Protein, Stimulatory Gs
- Gs G-Protein, Stimulatory
- Stimulatory Gs G-Protein
Below are MeSH descriptors whose meaning is more general than "GTP-Binding Protein alpha Subunits, Gs".
Below are MeSH descriptors whose meaning is more specific than "GTP-Binding Protein alpha Subunits, Gs".
This graph shows the total number of publications written about "GTP-Binding Protein alpha Subunits, Gs" by people in Harvard Catalyst Profiles by year, and whether "GTP-Binding Protein alpha Subunits, Gs" was a major or minor topic of these publication.
To see the data from this visualization as text,
click here.
| Year | Major Topic | Minor Topic | Total |
|---|
| 1996 | 0 | 1 | 1 |
| 1998 | 2 | 2 | 4 |
| 1999 | 0 | 1 | 1 |
| 2000 | 1 | 1 | 2 |
| 2001 | 4 | 1 | 5 |
| 2002 | 3 | 0 | 3 |
| 2003 | 2 | 1 | 3 |
| 2004 | 4 | 1 | 5 |
| 2005 | 3 | 0 | 3 |
| 2006 | 6 | 0 | 6 |
| 2007 | 5 | 0 | 5 |
| 2008 | 2 | 0 | 2 |
| 2009 | 6 | 3 | 9 |
| 2010 | 4 | 1 | 5 |
| 2011 | 6 | 0 | 6 |
| 2012 | 4 | 1 | 5 |
| 2013 | 6 | 1 | 7 |
| 2014 | 6 | 0 | 6 |
| 2015 | 7 | 1 | 8 |
| 2016 | 1 | 0 | 1 |
| 2017 | 8 | 3 | 11 |
| 2018 | 4 | 1 | 5 |
| 2019 | 3 | 1 | 4 |
| 2020 | 2 | 3 | 5 |
| 2021 | 2 | 2 | 4 |
| 2022 | 4 | 4 | 8 |
| 2023 | 0 | 3 | 3 |
| 2024 | 1 | 0 | 1 |
Below are the most recent publications written about "GTP-Binding Protein alpha Subunits, Gs" by people in Profiles.
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GNAS AS2 methylation status enables mechanism-based categorization of pseudohypoparathyroidism type 1B. JCI Insight. 2024 Mar 08; 9(5).
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Structural basis of amine odorant perception by a mammal olfactory receptor. Nature. 2023 Jun; 618(7963):193-200.
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The long-range interaction between two GNAS imprinting control regions delineates pseudohypoparathyroidism type 1B pathogenesis. J Clin Invest. 2023 04 17; 133(8).
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Multiplex Digital PCR Assay to Detect Multiple KRAS and GNAS Mutations Associated with Pancreatic Carcinogenesis from Minimal Specimen Amounts. J Mol Diagn. 2023 06; 25(6):367-377.
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Serine/Threonine Kinase 11 Plays a Canonical Role in Malignant Progression of KRAS -Mutant and GNAS -Wild-Type Intraductal Papillary Mucinous Neoplasms of the Pancreas. Ann Surg. 2023 02 01; 277(2):e384-e395.
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A naturally occurring membrane-anchored Gas variant, XLas, activates phospholipase Cß4. J Biol Chem. 2022 08; 298(8):102134.
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Lack of GNAS Remethylation During Oogenesis May Be a Cause of Sporadic Pseudohypoparathyroidism Type Ib. J Clin Endocrinol Metab. 2022 03 24; 107(4):e1610-e1619.
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Integrative RNA-omics Discovers GNAS Alternative Splicing as a Phenotypic Driver of Splicing Factor-Mutant Neoplasms. Cancer Discov. 2022 03 01; 12(3):836-855.
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Gnas Loss Causes Chondrocyte Fate Conversion in Cranial Suture Formation. J Dent Res. 2022 07; 101(8):931-941.
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Sporadic Pseudohypoparathyroidism Type 1B in Monozygotic Twins: Insights Into the Pathogenesis of Methylation Defects. J Clin Endocrinol Metab. 2022 02 17; 107(3):e947-e954.